Farnesol contributes to intestinal epithelial barrier function by enhancing tight junctions via the JAK/STAT3 signaling pathway in differentiated Caco-2 cells.

Fang, Yangxin; Wu, Chunrong; Wang, Qiuyue; Tang, Jianguo

Fang, Yangxin; Wu, Chunrong; Wang, Qiuyue; Tang, Jianguo.

Affiliation

Fang Y; Department of Trauma-Emergency & Critical Care Medicine Shanghai Fifth People's Hospital, Fudan University, Minhang district, 200240, Shanghai, China.
Wu C; Department of Trauma-Emergency & Critical Care Medicine Shanghai Fifth People's Hospital, Fudan University, Minhang district, 200240, Shanghai, China.
Wang Q; Department of Trauma-Emergency & Critical Care Medicine Shanghai Fifth People's Hospital, Fudan University, Minhang district, 200240, Shanghai, China.
Tang J; Department of Trauma-Emergency & Critical Care Medicine Shanghai Fifth People's Hospital, Fudan University, Minhang district, 200240, Shanghai, China. tangjianguo@5thhospital.com.

J Bioenerg Biomembr ; 51(6): 403-412, 2019 12.

Article in En | MEDLINE | ID: mdl-31845097

Subject(s)

Caco-2 Cells/metabolism; Epithelial Cells/metabolism; Farnesol/therapeutic use; Intestinal Mucosa/metabolism; STAT3 Transcription Factor/metabolism; Tight Junctions/metabolism; Cell Differentiation; Farnesol/pharmacology; Humans; Signal Transduction; Transfection

Key words

Farnesol; Intestinal epithelial barrier; Quorum-sensing molecule; STAT3; Tight junction

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Tight Junctions / Caco-2 Cells / Epithelial Cells / STAT3 Transcription Factor / Farnesol / Intestinal Mucosa Type of study: Prognostic_studies Limits: Humans Language: En Journal: J Bioenerg Biomembr Year: 2019 Document type: Article Affiliation country: China Country of publication: United States

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