Extracellular Acidosis Regulates the Expression of Inflammatory Mediators in Rat Epithelial Cells.

Riemann, A; Reime, S; Gießelmann, M; Thews, O

Riemann, A; Reime, S; Gießelmann, M; Thews, O.

Affiliation

Riemann A; Julius-Bernstein-Institute of Physiology, University of Halle, Halle (Saale), Germany. anne.riemann@medizin.uni-halle.de.
Reime S; Julius-Bernstein-Institute of Physiology, University of Halle, Halle (Saale), Germany.
Gießelmann M; Julius-Bernstein-Institute of Physiology, University of Halle, Halle (Saale), Germany.
Thews O; Julius-Bernstein-Institute of Physiology, University of Halle, Halle (Saale), Germany.

Adv Exp Med Biol ; 1232: 277-282, 2020.

Article in En | MEDLINE | ID: mdl-31893421

Subject(s)

Acidosis; Epithelial Cells; Gene Expression Regulation; Inflammation Mediators; p38 Mitogen-Activated Protein Kinases; Acidosis/metabolism; Animals; Cell Line; Chemokine CCL2/genetics; Cyclooxygenase 2/genetics; Epithelial Cells/metabolism; Inflammation Mediators/metabolism; MAP Kinase Signaling System/physiology; Nitric Oxide Synthase Type II/genetics; Rats; Tumor Necrosis Factor-alpha/genetics

Key words

Acidosis; MAP kinases; MCP-1; TNF-α; iNOS

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Acidosis / Gene Expression Regulation / Inflammation Mediators / P38 Mitogen-Activated Protein Kinases / Epithelial Cells Limits: Animals Language: En Journal: Adv Exp Med Biol Year: 2020 Document type: Article Affiliation country: Germany Country of publication: United States

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