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Sustained Increases in Cardiomyocyte Protein O-Linked ß-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment.
Ha, Chae-Myeong; Bakshi, Sayan; Brahma, Manoja K; Potter, Luke A; Chang, Samuel F; Sun, Zhihuan; Benavides, Gloria A; He, Lihao; Umbarkar, Prachi; Zou, Luyun; Curfman, Samuel; Sunny, Sini; Paterson, Andrew J; Rajasekaran, Namakkal-Soorappan; Barnes, Jarrod W; Zhang, Jianhua; Lal, Hind; Xie, Min; Darley-Usmar, Victor M; Chatham, John C; Wende, Adam R.
Affiliation
  • Ha CM; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Bakshi S; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Brahma MK; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Potter LA; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Chang SF; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Sun Z; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Benavides GA; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • He L; Division of Cardiovascular Disease, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Umbarkar P; Division of Cardiovascular Disease, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Zou L; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Curfman S; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Sunny S; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Paterson AJ; Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Rajasekaran NS; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Barnes JW; Division of Pulmonary, Allergy, and Critical Care Medicine, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Zhang J; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Lal H; Division of Cardiovascular Disease, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Xie M; Division of Cardiovascular Disease, Department of Medicine University of Alabama at Birmingham Birmingham AL.
  • Darley-Usmar VM; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Chatham JC; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
  • Wende AR; Division of Molecular and Cellular Pathology, Department of Pathology University of Alabama at Birmingham Birmingham AL.
J Am Heart Assoc ; 12(19): e029898, 2023 10 03.
Article in En | MEDLINE | ID: mdl-37750556
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cardiovascular Diseases / Myocytes, Cardiac Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Am Heart Assoc Year: 2023 Document type: Article Country of publication: United kingdom
Fulltext
Add to My VHL
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cardiovascular Diseases / Myocytes, Cardiac Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Am Heart Assoc Year: 2023 Document type: Article Country of publication: United kingdom