Reversion of v-H-ras-transformed NIH 3T3 cells by apigenin through inhibiting mitogen activated protein kinase and its downstream oncogenes.

Kuo, M L; Yang, N C

Kuo, M L; Yang, N C.

Affiliation

Kuo ML; Institute of Toxicology, College of Medicine, National Taiwan University, Taipei, Republic of China.

Biochem Biophys Res Commun ; 212(3): 767-75, 1995 Jul 26.

Article in En | MEDLINE | ID: mdl-7626110

Subject(s)

Flavonoids/pharmacology; Genes, ras; Oils, Volatile/pharmacology; 3T3 Cells; Animals; Calcium-Calmodulin-Dependent Protein Kinases/antagonists & inhibitors; Calcium-Calmodulin-Dependent Protein Kinases/genetics; Cell Division/drug effects; Chamomile; Genes, fos; Genes, jun; Genes, ras/drug effects; Mice; Oncogene Protein p21(ras)/genetics; Oncogenes/drug effects; Phenotype; Plants, Medicinal; RNA, Messenger/genetics; RNA, Messenger/metabolism; Transformation, Genetic/drug effects

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Collection: 01-internacional Database: MEDLINE Main subject: Flavonoids / Oils, Volatile / Genes, ras Limits: Animals Language: En Journal: Biochem Biophys Res Commun Year: 1995 Document type: Article Affiliation country: China Country of publication: United States

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