Extended survivability of prostate cancer cells in the absence of trophic factors: increased proliferation, evasion of apoptosis, and the role of apoptosis proteins.

Tang, D G; Li, L; Chopra, D P; Porter, A T

Tang, D G; Li, L; Chopra, D P; Porter, A T.

Affiliation

Tang DG; Department of Radiation Oncology, Wayne State University, Detroit, Michigan 48202, USA. dtang@sun.science.wayne.edu

Cancer Res ; 58(15): 3466-79, 1998 Aug 01.

Article in En | MEDLINE | ID: mdl-9699682

Subject(s)

Apoptosis/physiology; Neoplasm Proteins/physiology; Prostatic Neoplasms/pathology; Carrier Proteins/physiology; Cell Division/physiology; Cell Survival/physiology; Cyclin-Dependent Kinase Inhibitor p21; Cyclins/physiology; Humans; Male; Membrane Proteins/physiology; Prostate/cytology; Prostate/metabolism; Prostatic Hyperplasia/metabolism; Prostatic Hyperplasia/pathology; Prostatic Neoplasms/metabolism; Proto-Oncogene Proteins/physiology; Proto-Oncogene Proteins c-bcl-2/physiology; Tumor Suppressor Protein p53/physiology; bcl-2 Homologous Antagonist-Killer Protein; bcl-2-Associated X Protein; bcl-Associated Death Protein; bcl-X Protein

Search on Google

Add to My VHL

XML

PubMed Links

Collection: 01-internacional Database: MEDLINE Main subject: Prostatic Neoplasms / Apoptosis / Neoplasm Proteins Limits: Humans / Male Language: En Journal: Cancer Res Year: 1998 Document type: Article Affiliation country: United States Country of publication: United States

Search on Google

Add to My VHL

XML

PubMed Links