[The clinical pathophysiology of the early metabolic syndrome: pathogenetic roles of juvenile dyspituitarism, connective tissue dysplasiae and autoimmune thyroiditis].
Patol Fiziol Eksp Ter
; (3): 3-14, 2011.
Article
en Ru
| MEDLINE
| ID: mdl-22279733
ABSTRACT
A review of world literature, including authors' original papers, establishes the clinical pathophysiological hypothesis, initially coined in by authors in 2002, about regular transition of adolescent hypothalamic syndrome (obesity with rose striae) with age into early metabolic syndrome, complicated by autoimmune thyroiditis. Some evidences are obtained, that witness for marfanoid phenotype and chronic disequilibrium between local, autacoid-mediated and systemic, hormone-mediated regulation, typical for inherited connective tissue disorders, may promote this transition (6 fig., 3 tables, bibl. 92 ref.).
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Colección:
01-internacional
Base de datos:
MEDLINE
Asunto principal:
Enfermedades de la Hipófisis
/
Tiroiditis Autoinmune
/
Enfermedades del Tejido Conjuntivo
/
Síndrome Metabólico
Límite:
Adolescent
/
Humans
Idioma:
Ru
Revista:
Patol Fiziol Eksp Ter
Año:
2011
Tipo del documento:
Article