Aß-AGE aggravates cognitive deficit in rats via RAGE pathway.
Neuroscience
; 257: 1-10, 2014 Jan 17.
Article
en En
| MEDLINE
| ID: mdl-24188791
Palabras clave
AD; AGEs; AP; APP; Alzheimer's disease; Aß; Aß-AGE; DV; EDTA; EPSP; GSK3; HFS; LTP; MAPK; ML; NF-κB; PB; PCR; PS; PSD; RAGE; advanced glycation end-products; amyloid precursor protein; anterior to posterior; dorsal to ventral; ethylenediaminetetraacetic acid; excitatory postsynaptic potential; glycated Aß; glycation; glycogen synthase kinase 3; high-frequency stimulation; long-term potentiation; mid to lateral; mitogen-activated protein kinases; nuclear factor κB; pAb; phosphate buffer; polyclonal antibody; polymerase chain reaction; population spike; postsynaptic density; receptor for AGEs; receptor of advanced glycation endproducts; synthase kinase-3; ß-amyloid
Texto completo:
1
Colección:
01-internacional
Base de datos:
MEDLINE
Asunto principal:
Receptores Inmunológicos
/
Transducción de Señal
/
Péptidos beta-Amiloides
/
Trastornos del Conocimiento
Tipo de estudio:
Prognostic_studies
Límite:
Animals
Idioma:
En
Revista:
Neuroscience
Año:
2014
Tipo del documento:
Article
País de afiliación:
China
Pais de publicación:
Estados Unidos