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NRIP1/RIP140 siRNA-mediated attenuation counteracts mitochondrial dysfunction in Down syndrome.
Izzo, Antonella; Manco, Rosanna; Bonfiglio, Ferdinando; Calì, Gaetano; De Cristofaro, Tiziana; Patergnani, Simone; Cicatiello, Rita; Scrima, Rosella; Zannini, Mariastella; Pinton, Paolo; Conti, Anna; Nitsch, Lucio.
Afiliación
  • Izzo A; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Manco R; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Bonfiglio F; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Calì G; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • De Cristofaro T; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • Patergnani S; Department of Experimental and Diagnostic Medicine, University of Ferrara, Ferrara 44100, Italy.
  • Cicatiello R; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
  • Scrima R; Department of Clinical and Experimental Medicine, University of Foggia, Foggia 71100, Italy.
  • Zannini M; Institute of Experimental Endocrinology and Oncology, National Research Council, Naples 80131, Italy.
  • Pinton P; Department of Experimental and Diagnostic Medicine, University of Ferrara, Ferrara 44100, Italy.
  • Conti A; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy anconti@unina.it.
  • Nitsch L; Department of Molecular Medicine and Medical Biotechnology, University of Naples Federico II, Via Pansini 5, Naples 80131, Italy.
Hum Mol Genet ; 23(16): 4406-19, 2014 Aug 15.
Article en En | MEDLINE | ID: mdl-24698981
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Trisomía / Cromosomas Humanos Par 21 / Proteínas Nucleares / Síndrome de Down / Proteínas Adaptadoras Transductoras de Señales / Mitocondrias / Miocardio Tipo de estudio: Systematic_reviews Límite: Humans Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2014 Tipo del documento: Article País de afiliación: Italia Pais de publicación: Reino Unido
Texto completo
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XML
PubMed Links
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Trisomía / Cromosomas Humanos Par 21 / Proteínas Nucleares / Síndrome de Down / Proteínas Adaptadoras Transductoras de Señales / Mitocondrias / Miocardio Tipo de estudio: Systematic_reviews Límite: Humans Idioma: En Revista: Hum Mol Genet Asunto de la revista: BIOLOGIA MOLECULAR / GENETICA MEDICA Año: 2014 Tipo del documento: Article País de afiliación: Italia Pais de publicación: Reino Unido