Your browser doesn't support javascript.
loading
Excessive transforming growth factor-ß signaling is a common mechanism in osteogenesis imperfecta.
Grafe, Ingo; Yang, Tao; Alexander, Stefanie; Homan, Erica P; Lietman, Caressa; Jiang, Ming Ming; Bertin, Terry; Munivez, Elda; Chen, Yuqing; Dawson, Brian; Ishikawa, Yoshihiro; Weis, Mary Ann; Sampath, T Kuber; Ambrose, Catherine; Eyre, David; Bächinger, Hans Peter; Lee, Brendan.
Afiliación
  • Grafe I; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Yang T; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Alexander S; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Homan EP; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Lietman C; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Jiang MM; 1] Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA. [2] Howard Hughes Medical Institute, Houston, Texas, USA.
  • Bertin T; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Munivez E; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Chen Y; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA.
  • Dawson B; 1] Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA. [2] Howard Hughes Medical Institute, Houston, Texas, USA.
  • Ishikawa Y; 1] Research Department, Shriners Hospital for Children, Portland, Oregon, USA. [2] Department of Biochemistry and Molecular Biology, Oregon Health and Science University, Portland, Oregon, USA.
  • Weis MA; Department of Orthopaedics and Sports Medicine, University of Washington, Seattle, Washington, USA.
  • Sampath TK; Genzyme Research Center, Framingham, Massachusetts, USA.
  • Ambrose C; Department of Orthopaedic Surgery, University of Texas Health Science Center at Houston, Houston, Texas, USA.
  • Eyre D; Department of Orthopaedics and Sports Medicine, University of Washington, Seattle, Washington, USA.
  • Bächinger HP; 1] Research Department, Shriners Hospital for Children, Portland, Oregon, USA. [2] Department of Biochemistry and Molecular Biology, Oregon Health and Science University, Portland, Oregon, USA.
  • Lee B; 1] Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA. [2] Howard Hughes Medical Institute, Houston, Texas, USA.
Nat Med ; 20(6): 670-5, 2014 Jun.
Article en En | MEDLINE | ID: mdl-24793237
Asunto(s)
Texto completo
Añadir a Mi BVS
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Osteogénesis Imperfecta / Transducción de Señal / Factor de Crecimiento Transformador beta Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos
Texto completo
Añadir a Mi BVS
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Osteogénesis Imperfecta / Transducción de Señal / Factor de Crecimiento Transformador beta Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Nat Med Asunto de la revista: BIOLOGIA MOLECULAR / MEDICINA Año: 2014 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Estados Unidos