Mechanisms of action of therapeutic amyloidogenic hexapeptides in amelioration of inflammatory brain disease.

Kurnellas, Michael P; Schartner, Jill M; Fathman, C Garrison; Jagger, Ann; Steinman, Lawrence; Rothbard, Jonathan B

Kurnellas, Michael P; Schartner, Jill M; Fathman, C Garrison; Jagger, Ann; Steinman, Lawrence; Rothbard, Jonathan B.

Afiliación

Kurnellas MP; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Schartner JM; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Fathman CG; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Jagger A; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305.
Steinman L; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305 steinman@stanford.edu.
Rothbard JB; Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicine, Stanford, CA 94305 Department of Neurology and Neurological Sciences and Department of Medicine, Division of Immunology, Stanford University School of Medicin

J Exp Med ; 211(9): 1847-56, 2014 Aug 25.

Article en En | MEDLINE | ID: mdl-25073790

Asunto(s)

Amiloide/inmunología; Amiloide/uso terapéutico; Encefalomielitis Autoinmune Experimental/inmunología; Encefalomielitis Autoinmune Experimental/terapia; Fragmentos de Péptidos/inmunología; Fragmentos de Péptidos/uso terapéutico; Traslado Adoptivo; Adulto; Animales; Encefalomielitis Autoinmune Experimental/genética; Femenino; Expresión Génica; Humanos; Inmunosupresores/uso terapéutico; Interferón Tipo I/metabolismo; Interferón gamma/metabolismo; Interleucina-6/metabolismo; Ratones; Ratones Endogámicos C57BL; Ratones Noqueados; Células TH1/inmunología; Células Th17/inmunología; Factor de Necrosis Tumoral alfa/metabolismo; Proteínas tau/inmunología; Proteínas tau/uso terapéutico

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Fragmentos de Péptidos / Encefalomielitis Autoinmune Experimental / Amiloide Límite: Adult / Animals / Female / Humans Idioma: En Revista: J Exp Med Año: 2014 Tipo del documento: Article Pais de publicación: Estados Unidos

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