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Sodium selenate retards epileptogenesis in acquired epilepsy models reversing changes in protein phosphatase 2A and hyperphosphorylated tau.
Liu, Shi-Jie; Zheng, Ping; Wright, David K; Dezsi, Gabi; Braine, Emma; Nguyen, Thanh; Corcoran, Niall M; Johnston, Leigh A; Hovens, Christopher M; Mayo, Jamie N; Hudson, Matthew; Shultz, Sandy R; Jones, Nigel C; O'Brien, Terence J.
Afiliación
  • Liu SJ; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Zheng P; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Wright DK; 2 The Florey Institute of Neuroscience and Mental Health, Parkville, VIC, Australia 3 Department of Anatomy and Neuroscience, The University of Melbourne, Parkville, VIC, Australia.
  • Dezsi G; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Braine E; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Nguyen T; 4 Department of Surgery, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Corcoran NM; 4 Department of Surgery, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Johnston LA; 2 The Florey Institute of Neuroscience and Mental Health, Parkville, VIC, Australia 5 Department of Electrical and Electronic Engineering, The University of Melbourne, Parkville, VIC, Australia.
  • Hovens CM; 4 Department of Surgery, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Mayo JN; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Hudson M; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia.
  • Shultz SR; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia sshultz@unimelb.edu.au ncjones@unimelb.edu.au obrientj@unimelb.edu.au.
  • Jones NC; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia sshultz@unimelb.edu.au ncjones@unimelb.edu.au obrientj@unimelb.edu.au.
  • O'Brien TJ; 1 Department of Medicine, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia 6 Department of Neurology, Melbourne Brain Centre, The Royal Melbourne Hospital, The University of Melbourne, Parkville, VIC, Australia sshultz@unimelb.edu.au ncjone
Brain ; 139(Pt 7): 1919-38, 2016 07.
Article en En | MEDLINE | ID: mdl-27289302

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Proteínas tau / Modelos Animales de Enfermedad / Epilepsia / Proteína Fosfatasa 2 / Ácido Selénico / Anticonvulsivantes Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Brain Año: 2016 Tipo del documento: Article País de afiliación: Australia

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Encéfalo / Proteínas tau / Modelos Animales de Enfermedad / Epilepsia / Proteína Fosfatasa 2 / Ácido Selénico / Anticonvulsivantes Tipo de estudio: Etiology_studies Límite: Animals Idioma: En Revista: Brain Año: 2016 Tipo del documento: Article País de afiliación: Australia