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Suppression of MAPK11 or HIPK3 reduces mutant Huntingtin levels in Huntington's disease models.
Yu, Meng; Fu, Yuhua; Liang, Yijiang; Song, Haikun; Yao, Yao; Wu, Peng; Yao, Yuwei; Pan, Yuyin; Wen, Xue; Ma, Lixiang; Hexige, Saiyin; Ding, Yu; Luo, Shouqing; Lu, Boxun.
Afiliación
  • Yu M; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Fu Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Liang Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Song H; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Yao Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Wu P; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Yao Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Pan Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Wen X; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Ma L; Department of Anatomy and Histology & Embryology, Shanghai Medical College, Fudan University, Shanghai 200032, China.
  • Hexige S; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Ding Y; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
  • Luo S; Peninsula Schools of Medicine and Dentistry, Institute of Translational and Stratified Medicine, University of Plymouth, Research Way, Plymouth, PL68BU, UK.
  • Lu B; State Key Laboratory of Medical Neurobiology, Huashan Hospital, School of Life Sciences, Collaborative Innovation Center for Genetics and Development, Fudan University, Shanghai 200438, China.
Cell Res ; 27(12): 1441-1465, 2017 Dec.
Article en En | MEDLINE | ID: mdl-29151587
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Serina-Treonina Quinasas / Enfermedad de Huntington / Péptidos y Proteínas de Señalización Intracelular / Proteína Quinasa 11 Activada por Mitógenos / Modelos Animales de Enfermedad / Proteína Huntingtina / Mutación Límite: Animals / Humans Idioma: En Revista: Cell Res Año: 2017 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Proteínas Serina-Treonina Quinasas / Enfermedad de Huntington / Péptidos y Proteínas de Señalización Intracelular / Proteína Quinasa 11 Activada por Mitógenos / Modelos Animales de Enfermedad / Proteína Huntingtina / Mutación Límite: Animals / Humans Idioma: En Revista: Cell Res Año: 2017 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido