NMT1 inhibition modulates breast cancer progression through stress-triggered JNK pathway.

Deng, Lu; Gao, Xinlei; Liu, Bingjie; He, Xueyan; Xu, Jiahui; Qiang, Jiankun; Wu, Qingfa; Liu, Suling

Deng, Lu; Gao, Xinlei; Liu, Bingjie; He, Xueyan; Xu, Jiahui; Qiang, Jiankun; Wu, Qingfa; Liu, Suling.

Afiliación

Deng L; Fudan University Shanghai Cancer Center and Institutes of Biomedical Sciences; Shanghai Medical College; Key Laboratory of Breast Cancer in Shanghai; Innovation Center for Cell Signaling Network; Cancer Institutes, Fudan University, 200032, Shanghai, China.
Gao X; School of Life Science, The CAS Key Laboratory of Innate Immunity and Chronic Disease, University of Science and Technology of China, 230027, Hefei, Anhui, China.
Liu B; School of Life Science, The CAS Key Laboratory of Innate Immunity and Chronic Disease, University of Science and Technology of China, 230027, Hefei, Anhui, China.
He X; Fudan University Shanghai Cancer Center and Institutes of Biomedical Sciences; Shanghai Medical College; Key Laboratory of Breast Cancer in Shanghai; Innovation Center for Cell Signaling Network; Cancer Institutes, Fudan University, 200032, Shanghai, China.
Xu J; School of Life Science, The CAS Key Laboratory of Innate Immunity and Chronic Disease, University of Science and Technology of China, 230027, Hefei, Anhui, China.
Qiang J; Fudan University Shanghai Cancer Center and Institutes of Biomedical Sciences; Shanghai Medical College; Key Laboratory of Breast Cancer in Shanghai; Innovation Center for Cell Signaling Network; Cancer Institutes, Fudan University, 200032, Shanghai, China.
Wu Q; Fudan University Shanghai Cancer Center and Institutes of Biomedical Sciences; Shanghai Medical College; Key Laboratory of Breast Cancer in Shanghai; Innovation Center for Cell Signaling Network; Cancer Institutes, Fudan University, 200032, Shanghai, China.
Liu S; Fudan University Shanghai Cancer Center and Institutes of Biomedical Sciences; Shanghai Medical College; Key Laboratory of Breast Cancer in Shanghai; Innovation Center for Cell Signaling Network; Cancer Institutes, Fudan University, 200032, Shanghai, China.

Cell Death Dis ; 9(12): 1143, 2018 11 16.

Article en En | MEDLINE | ID: mdl-30446635

Asunto(s)

Aciltransferasas/genética; Estrés del Retículo Endoplásmico/efectos de los fármacos; Ácido Mirístico/farmacología; Neoplasias de la Mama Triple Negativas/tratamiento farmacológico; Animales; Autofagia/efectos de los fármacos; Línea Celular Tumoral; Proliferación Celular/efectos de los fármacos; Progresión de la Enfermedad; Estrés del Retículo Endoplásmico/genética; Femenino; Regulación Neoplásica de la Expresión Génica; Humanos; Sistema de Señalización de MAP Quinasas/efectos de los fármacos; Ratones; Invasividad Neoplásica/genética; Invasividad Neoplásica/patología; Procesamiento Proteico-Postraduccional/efectos de los fármacos; Neoplasias de la Mama Triple Negativas/genética; Neoplasias de la Mama Triple Negativas/patología; Ensayos Antitumor por Modelo de Xenoinjerto

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Aciltransferasas / Ácido Mirístico / Estrés del Retículo Endoplásmico / Neoplasias de la Mama Triple Negativas Límite: Animals / Female / Humans Idioma: En Revista: Cell Death Dis Año: 2018 Tipo del documento: Article País de afiliación: China Pais de publicación: Reino Unido

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