Consequences of Making the Inactive Active Through Changes in Antisense Oligonucleotide Chemistries.

Zaw, Khine; Greer, Kane; Aung-Htut, May Thandar; Mitrpant, Chalermchai; Veedu, Rakesh N; Fletcher, Sue; Wilton, Steve D

Zaw, Khine; Greer, Kane; Aung-Htut, May Thandar; Mitrpant, Chalermchai; Veedu, Rakesh N; Fletcher, Sue; Wilton, Steve D.

Afiliación

Zaw K; Centre for Molecular Medicine and Innovative Therapeutics, Murdoch University, Perth, WA, Australia.
Greer K; Department of Biochemistry, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand.
Aung-Htut MT; Centre for Molecular Medicine and Innovative Therapeutics, Murdoch University, Perth, WA, Australia.
Mitrpant C; Perron Institute for Neurological and Translational Science and Centre for Neuromuscular and Neurological Disorders, The University of Western Australia, Perth, WA, Australia.
Veedu RN; Centre for Molecular Medicine and Innovative Therapeutics, Murdoch University, Perth, WA, Australia.
Fletcher S; Perron Institute for Neurological and Translational Science and Centre for Neuromuscular and Neurological Disorders, The University of Western Australia, Perth, WA, Australia.
Wilton SD; Department of Biochemistry, Faculty of Medicine Siriraj Hospital, Mahidol University, Bangkok, Thailand.

Front Genet ; 10: 1249, 2019.

Article en En | MEDLINE | ID: mdl-31956327

Palabras clave

DMD; antisense oligonucleotide; cryptic splice site; locked nucleic acid; locked nucleic acid/2'-O-methyl mixmer

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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Idioma: En Revista: Front Genet Año: 2019 Tipo del documento: Article País de afiliación: Australia Pais de publicación: Suiza

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