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Ins1-Cre and Ins1-CreER Gene Replacement Alleles Are Susceptible To Silencing By DNA Hypermethylation.
Mosleh, Elham; Ou, Kristy; Haemmerle, Matthew W; Tembo, Teguru; Yuhas, Andrew; Carboneau, Bethany A; Townsend, Shannon E; Bosma, Karin J; Gannon, Maureen; O'Brien, Richard M; Stoffers, Doris A; Golson, Maria L.
Afiliación
  • Mosleh E; Department of Genetics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Ou K; Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Haemmerle MW; Department of Genetics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Tembo T; Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Yuhas A; Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Carboneau BA; Department of Genetics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Townsend SE; Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Bosma KJ; Department of Genetics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • Gannon M; Institute for Diabetes, Obesity, and Metabolism, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.
  • O'Brien RM; Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee.
  • Stoffers DA; Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee.
  • Golson ML; Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee.
Endocrinology ; 161(8)2020 08 01.
Article en En | MEDLINE | ID: mdl-32267917
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Recombinación Genética / Integrasas / Metilación de ADN / Células Secretoras de Insulina / Insulina Límite: Animals / Female / Humans / Male Idioma: En Revista: Endocrinology Año: 2020 Tipo del documento: Article Pais de publicación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Recombinación Genética / Integrasas / Metilación de ADN / Células Secretoras de Insulina / Insulina Límite: Animals / Female / Humans / Male Idioma: En Revista: Endocrinology Año: 2020 Tipo del documento: Article Pais de publicación: Estados Unidos