Your browser doesn't support javascript.
loading
Expression of mutant exon 1 huntingtin fragments in human neural stem cells and neurons causes inclusion formation and mitochondrial dysfunction.
Ghosh, Rhia; Wood-Kaczmar, Alison; Dobson, Lucianne; Smith, Edward J; Sirinathsinghji, Eva C; Kriston-Vizi, Janos; Hargreaves, Iain P; Heaton, Robert; Herrmann, Frank; Abramov, Andrey Y; Lam, Amanda J; Heales, Simon J; Ketteler, Robin; Bates, Gillian P; Andre, Ralph; Tabrizi, Sarah J.
Afiliación
  • Ghosh R; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Wood-Kaczmar A; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Dobson L; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Smith EJ; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Sirinathsinghji EC; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Kriston-Vizi J; MRC Laboratory for Molecular Cell Biology, University College London, London, UK.
  • Hargreaves IP; School of Pharmacy, Liverpool John Moores University, Liverpool, UK.
  • Heaton R; School of Pharmacy, Liverpool John Moores University, Liverpool, UK.
  • Herrmann F; Evotec AG, Hamburg, Germany.
  • Abramov AY; Department of Clinical and Movement Neurosciences, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Lam AJ; Neurometabolic Unit, National Hospital for Neurology and Neurosurgery, London, UK.
  • Heales SJ; Neurometabolic Unit, National Hospital for Neurology and Neurosurgery, London, UK.
  • Ketteler R; MRC Laboratory for Molecular Cell Biology, University College London, London, UK.
  • Bates GP; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Andre R; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
  • Tabrizi SJ; Huntington's Disease Centre, Department of Neurodegenerative Disease, UCL Queen Square Institute of Neurology, University College London, London, UK.
FASEB J ; 34(6): 8139-8154, 2020 06.
Article en En | MEDLINE | ID: mdl-32329133
Asunto(s)
Palabras clave
Texto completo
Añadir a Mi BVS
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Cuerpos de Inclusión / Células-Madre Neurales / Proteína Huntingtina / Mitocondrias / Neuronas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: FASEB J Asunto de la revista: BIOLOGIA / FISIOLOGIA Año: 2020 Tipo del documento: Article País de afiliación: Reino Unido
Texto completo
Añadir a Mi BVS
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Cuerpos de Inclusión / Células-Madre Neurales / Proteína Huntingtina / Mitocondrias / Neuronas Tipo de estudio: Etiology_studies / Prognostic_studies Límite: Humans Idioma: En Revista: FASEB J Asunto de la revista: BIOLOGIA / FISIOLOGIA Año: 2020 Tipo del documento: Article País de afiliación: Reino Unido