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Enhancer reprogramming driven by high-order assemblies of transcription factors promotes phenotypic plasticity and breast cancer endocrine resistance.
Bi, Mingjun; Zhang, Zhao; Jiang, Yi-Zhou; Xue, Pengya; Wang, Hu; Lai, Zhao; Fu, Xiaoyong; De Angelis, Carmine; Gong, Yue; Gao, Zhen; Ruan, Jianhua; Jin, Victor X; Marangoni, Elisabetta; Montaudon, Elodie; Glass, Christopher K; Li, Wei; Huang, Tim Hui-Ming; Shao, Zhi-Ming; Schiff, Rachel; Chen, Lizhen; Liu, Zhijie.
Afiliación
  • Bi M; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Zhang Z; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Jiang YZ; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Xue P; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Wang H; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Lai Z; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Fu X; Department of Medicine, Department of Molecular and Cellular Biology, Lester & Sue Smith Breast Center, Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX, USA.
  • De Angelis C; Department of Medicine, Department of Molecular and Cellular Biology, Lester & Sue Smith Breast Center, Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX, USA.
  • Gong Y; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Gao Z; Department of Computer Science, University of Texas at San Antonio, San Antonio, TX, USA.
  • Ruan J; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Jin VX; Department of Computer Science, University of Texas at San Antonio, San Antonio, TX, USA.
  • Marangoni E; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Montaudon E; Translational Research Department, Institut Curie, PSL University, Paris, France.
  • Glass CK; Translational Research Department, Institut Curie, PSL University, Paris, France.
  • Li W; Department of Cellular and Molecular Medicine, Institute of Genomic Medicine, University of California at San Diego, La Jolla, CA, USA.
  • Huang TH; Department of Biological Chemistry, University of California, Irvine, Irvine, CA, USA.
  • Shao ZM; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA.
  • Schiff R; Department of Breast Surgery, Fudan University Shanghai Cancer Center, Shanghai, China.
  • Chen L; Department of Medicine, Department of Molecular and Cellular Biology, Lester & Sue Smith Breast Center, Dan L. Duncan Comprehensive Cancer Center, Baylor College of Medicine, Houston, TX, USA.
  • Liu Z; Department of Molecular Medicine, Mays Cancer Center, University of Texas Health Science Center at San Antonio, San Antonio, TX, USA. ChenL7@uthscsa.edu.
Nat Cell Biol ; 22(6): 701-715, 2020 06.
Article en En | MEDLINE | ID: mdl-32424275
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Adaptación Fisiológica / Regulación Neoplásica de la Expresión Génica / Factor de Transcripción AP-1 / Resistencia a Antineoplásicos / Factor de Transcripción GATA3 / Reprogramación Celular Límite: Animals / Female / Humans Idioma: En Revista: Nat Cell Biol Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido
Texto completo
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Texto completo: 1 Colección: 01-internacional Base de datos: MEDLINE Asunto principal: Neoplasias de la Mama / Adaptación Fisiológica / Regulación Neoplásica de la Expresión Génica / Factor de Transcripción AP-1 / Resistencia a Antineoplásicos / Factor de Transcripción GATA3 / Reprogramación Celular Límite: Animals / Female / Humans Idioma: En Revista: Nat Cell Biol Año: 2020 Tipo del documento: Article País de afiliación: Estados Unidos Pais de publicación: Reino Unido