Your browser doesn't support javascript.
loading
STING: Stay near to STIM(1) neuroprotection.
Lee, Hong-Gyun; Quintana, Francisco J.
Affiliation
  • Lee HG; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
  • Quintana FJ; Ann Romney Center for Neurologic Diseases, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA; Broad Institute of MIT and Harvard, Cambridge, MA, USA; Gene Lay Institute of Immunology and Inflammation, Boston, MA, USA. Electronic address: fquintana@rics.bwh.harvard.edu.
Mol Cell ; 84(14): 2596-2597, 2024 07 25.
Article in En | MEDLINE | ID: mdl-39059368
ABSTRACT
In a recent publication in Cell, Woo et al.1 report that stimulator of interferon genes (STING) links inflammation with glutamate-driven excitotoxicity to induce ferroptosis, identifying a mechanism of inflammation-induced neurodegeneration and also a novel candidate therapeutic target for multiple sclerosis.
Subject(s)
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Neuroprotection / Ferroptosis / Membrane Proteins / Multiple Sclerosis Limits: Animals / Humans Language: En Journal: Mol Cell Journal subject: BIOLOGIA MOLECULAR Year: 2024 Document type: Article Affiliation country: Country of publication:
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Neuroprotection / Ferroptosis / Membrane Proteins / Multiple Sclerosis Limits: Animals / Humans Language: En Journal: Mol Cell Journal subject: BIOLOGIA MOLECULAR Year: 2024 Document type: Article Affiliation country: Country of publication: