Inhibition of autophagy and glycolysis by nitric oxide during hypoxia-reoxygenation impairs cellular bioenergetics and promotes cell death in primary neurons.
Free Radic Biol Med
; 65: 1215-1228, 2013 Dec.
Article
de En
| MEDLINE
| ID: mdl-24056030
Mots clés
2-DG; 2-deoxyglucose; 3-MA; 3-methyladenine; CQ; DetaNO, (Z)-1-[N-(2-aminoethyl)-N-(2-ammonioethyl)amino]diazen-1-ium-1,2-diolate; DetaNONOate; ECAR; FCCP; Free radicals; Glucose; LC3; Mitochondria; NOS; OCR; carbonyl cyanide 4-(trifluoromethoxy)phenylhydrazone; chloroquine; extracellular acidification rate; microtubule-associated protein light-chain 3; nitric oxide synthase; oxygen consumption rate
Texte intégral:
1
Collection:
01-internacional
Base de données:
MEDLINE
Sujet principal:
Autophagie
/
Métabolisme énergétique
/
Glycolyse
/
Neurones
/
Monoxyde d'azote
Limites:
Animals
Langue:
En
Journal:
Free Radic Biol Med
Sujet du journal:
BIOQUIMICA
/
MEDICINA
Année:
2013
Type de document:
Article
Pays d'affiliation:
États-Unis d'Amérique
Pays de publication:
États-Unis d'Amérique