Prokineticin signaling is required for the maintenance of a de novo population of c-KITâº cells to sustain neuroblastoma progression.

Lau, S-T; Hansford, L M; Chan, W-K; Chan, G C-F; Wan, T S-K; Wong, K K-Y; Kaplan, D R; Tam, P K-H; Ngan, E S-W

Prokineticin signaling is required for the maintenance of a de novo population of c-KITâº cells to sustain neuroblastoma progression.

Lau, S-T; Hansford, L M; Chan, W-K; Chan, G C-F; Wan, T S-K; Wong, K K-Y; Kaplan, D R; Tam, P K-H; Ngan, E S-W.

Affiliation

Lau ST; Department of Surgery, University of Hong Kong, Hong Kong SAR, China.
Hansford LM; Department of Molecular and Medical Genetics, University of Toronto, Toronto Medical Discovery Towers, Toronto, Ontario, Canada.
Chan WK; Department of Paediatrics and Adolescent Medicine, University of Hong Kong, Hong Kong SAR, China.
Chan GC; Department of Paediatrics and Adolescent Medicine, University of Hong Kong, Hong Kong SAR, China.
Wan TS; Department of Pathology, University of Hong Kong, Hong Kong SAR, China.
Wong KK; Department of Surgery, University of Hong Kong, Hong Kong SAR, China.
Kaplan DR; Department of Molecular and Medical Genetics, University of Toronto, Toronto Medical Discovery Towers, Toronto, Ontario, Canada.
Tam PK; 1] Department of Surgery, University of Hong Kong, Hong Kong SAR, China [2] Centre for Reproduction, Development and Growth, University of Hong Kong, Hong Kong SAR, China.
Ngan ES; 1] Department of Surgery, University of Hong Kong, Hong Kong SAR, China [2] Centre for Reproduction, Development and Growth, University of Hong Kong, Hong Kong SAR, China.

Oncogene ; 34(8): 1019-34, 2015 Feb 19.

Article de En | MEDLINE | ID: mdl-24632619

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Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Cellules souches tumorales / Protéines proto-oncogènes c-kit / Facteur de croissance endothéliale vasculaire EG-VEGF / Neuroblastome Limites: Animals / Humans / Male Langue: En Journal: Oncogene Sujet du journal: BIOLOGIA MOLECULAR / NEOPLASIAS Année: 2015 Type de document: Article Pays d'affiliation: Chine Pays de publication: Royaume-Uni

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