Melatonin as a therapy in cardiac ischemia-reperfusion injury: Potential mechanisms by which MT2 activation mediates cardioprotection.

Singhanat, Kodchanan; Apaijai, Nattayaporn; Jaiwongkam, Thidarat; Kerdphoo, Sasiwan; Chattipakorn, Siriporn C; Chattipakorn, Nipon

Singhanat, Kodchanan; Apaijai, Nattayaporn; Jaiwongkam, Thidarat; Kerdphoo, Sasiwan; Chattipakorn, Siriporn C; Chattipakorn, Nipon.

Affiliation

Singhanat K; Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.
Apaijai N; Center of Excellence in Cardiac Electrophysiology Research, Chiang Mai University, Chiang Mai 50200, Thailand.
Jaiwongkam T; Cardiac Electrophysiology Unit, Department of Physiology, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.
Kerdphoo S; Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.
Chattipakorn SC; Center of Excellence in Cardiac Electrophysiology Research, Chiang Mai University, Chiang Mai 50200, Thailand.
Chattipakorn N; Cardiac Electrophysiology Research and Training Center, Faculty of Medicine, Chiang Mai University, Chiang Mai 50200, Thailand.

J Adv Res ; 29: 33-44, 2021 03.

Article de En | MEDLINE | ID: mdl-33842003

Sujet(s)
Mots clés

Cardiac I/R injury; Cardiomyocyte death; Left ventricular function; Melatonin; Melatonin receptors; Mitochondria

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Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Cardiotoniques / Lésion de reperfusion myocardique / Récepteur de la mélatonine de type MT2 / Mélatonine Limites: Animals Langue: En Journal: J Adv Res Année: 2021 Type de document: Article Pays d'affiliation: Thaïlande

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