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DnaJ-induced TLR7 mediates an increase in interferons through the TLR4-engaged AKT/NF-κB and JNK signaling pathways in macrophages.
Yu, Hyeonseung; Huh, Jin-Won; Bai, Fang; Ha, Un-Hwan.
Affiliation
  • Yu H; Department of Biotechnology and Bioinformatics, Korea University, Sejong, 30019, Republic of Korea; Interdisciplinary Graduate Program for Artificial Intelligence Smart Convergence Technology, Korea University, Sejong, 30019, Republic of Korea.
  • Huh JW; Department of Biotechnology and Bioinformatics, Korea University, Sejong, 30019, Republic of Korea.
  • Bai F; State Key Laboratory of Medicinal Chemical Biology, Key Laboratory of Molecular Microbiology and Technology of the Ministry of Education, Department of Microbiology, Nankai University, Tianjin, 300071, China.
  • Ha UH; Department of Biotechnology and Bioinformatics, Korea University, Sejong, 30019, Republic of Korea; Interdisciplinary Graduate Program for Artificial Intelligence Smart Convergence Technology, Korea University, Sejong, 30019, Republic of Korea. Electronic address: haunhwan@korea.ac.kr.
Microb Pathog ; 165: 105465, 2022 Apr.
Article de En | MEDLINE | ID: mdl-35247500

Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Facteur de transcription NF-kappa B / Récepteur de type Toll-7 Type d'étude: Prognostic_studies Langue: En Journal: Microb Pathog Sujet du journal: DOENCAS TRANSMISSIVEIS / MICROBIOLOGIA Année: 2022 Type de document: Article Pays de publication: Royaume-Uni

Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Facteur de transcription NF-kappa B / Récepteur de type Toll-7 Type d'étude: Prognostic_studies Langue: En Journal: Microb Pathog Sujet du journal: DOENCAS TRANSMISSIVEIS / MICROBIOLOGIA Année: 2022 Type de document: Article Pays de publication: Royaume-Uni