Hepatocyte-specific depletion of Nnmt protects mice from non-alcoholic steatohepatitis.

Li, Dandan; Yi, Chuanyou; Huang, He; Li, Jin; Hong, Shangyu

Li, Dandan; Yi, Chuanyou; Huang, He; Li, Jin; Hong, Shangyu.

Affiliation

Li D; State Key Laboratory of Genetic Engineering, School of Life Sciences, Human Phenome Institute, and Institute of Metabolism and Integrative Biology, Fudan University, Shanghai, 200438, China.
Yi C; State Key Laboratory of Genetic Engineering, School of Life Sciences, Human Phenome Institute, and Institute of Metabolism and Integrative Biology, Fudan University, Shanghai, 200438, China.
Huang H; State Key Laboratory of Genetic Engineering, School of Life Sciences, Human Phenome Institute, and Institute of Metabolism and Integrative Biology, Fudan University, Shanghai, 200438, China.
Li J; State Key Laboratory of Genetic Engineering, School of Life Sciences, Human Phenome Institute, and Institute of Metabolism and Integrative Biology, Fudan University, Shanghai, 200438, China. Electronic address: li_jin_lifescience@fudan.edu.cn.
Hong S; State Key Laboratory of Genetic Engineering, School of Life Sciences, Human Phenome Institute, and Institute of Metabolism and Integrative Biology, Fudan University, Shanghai, 200438, China. Electronic address: shangyu_hong@fudan.edu.cn.

J Hepatol ; 77(3): 882-884, 2022 09.

Article de En | MEDLINE | ID: mdl-35367283

Sujet(s)
Mots clés

conditional KO; liver damage; unfolded protein response

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Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Stéatose hépatique non alcoolique Type d'étude: Etiology_studies Limites: Animals Langue: En Journal: J Hepatol Sujet du journal: GASTROENTEROLOGIA Année: 2022 Type de document: Article Pays d'affiliation: Chine Pays de publication: Pays-Bas

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