Deleterious effects of cardiomyocyte-specific prostaglandin E2 EP3 receptor overexpression on cardiac function after myocardial infarction.

Maxwell, DruAnne L; Bryson, Timothy D; Taube, David; Xu, Jiang; Peterson, Edward; Harding, Pamela

Maxwell, DruAnne L; Bryson, Timothy D; Taube, David; Xu, Jiang; Peterson, Edward; Harding, Pamela.

Affiliation

Maxwell DL; Department of Physiology, Wayne State University School of Medicine, USA; Hypertension and Vascular Research Division, Department of Internal Medicine, Henry Ford Health, Detroit, MI, USA.
Bryson TD; Hypertension and Vascular Research Division, Department of Internal Medicine, Henry Ford Health, Detroit, MI, USA.
Taube D; Hypertension and Vascular Research Division, Department of Internal Medicine, Henry Ford Health, Detroit, MI, USA.
Xu J; Hypertension and Vascular Research Division, Department of Internal Medicine, Henry Ford Health, Detroit, MI, USA.
Peterson E; Department of Public Health Sciences, Henry Ford Health, Detroit, MI, USA.
Harding P; Department of Physiology, Wayne State University School of Medicine, USA; Hypertension and Vascular Research Division, Department of Internal Medicine, Henry Ford Health, Detroit, MI, USA. Electronic address: phardin1@hfhs.org.

Life Sci ; 313: 121277, 2023 Jan 15.

Article de En | MEDLINE | ID: mdl-36521546

Sujet(s)
Mots clés

EP3; Myocardial infarction; Prostaglandin E2

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Texte intégral: 1 Collection: 01-internacional Base de données: MEDLINE Sujet principal: Myocytes cardiaques / Sous-type EP3 des récepteurs des prostaglandines E / Infarctus du myocarde Type d'étude: Prognostic_studies Limites: Animals / Humans Langue: En Journal: Life Sci Année: 2023 Type de document: Article Pays d'affiliation: États-Unis d'Amérique Pays de publication: Pays-Bas

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