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Hypoxia activates a platelet-derived growth factor receptor/phosphatidylinositol 3-kinase/Akt pathway that results in glycogen synthase kinase-3 inactivation.
Chen, E Y; Mazure, N M; Cooper, J A; Giaccia, A J.
Affiliation
  • Chen EY; Department of Radiation Oncology, Center for Clinical Science Research-South, Stanford, California 94305-5152, USA.
Cancer Res ; 61(6): 2429-33, 2001 Mar 15.
Article in En | MEDLINE | ID: mdl-11289110
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Collection: 01-internacional Database: MEDLINE Main subject: Oxygen / Signal Transduction / Proto-Oncogene Proteins / Protein Serine-Threonine Kinases / Calcium-Calmodulin-Dependent Protein Kinases / Phosphatidylinositol 3-Kinases / Receptor, Platelet-Derived Growth Factor beta Limits: Animals / Humans Language: En Journal: Cancer Res Year: 2001 Document type: Article Affiliation country: Country of publication:
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Collection: 01-internacional Database: MEDLINE Main subject: Oxygen / Signal Transduction / Proto-Oncogene Proteins / Protein Serine-Threonine Kinases / Calcium-Calmodulin-Dependent Protein Kinases / Phosphatidylinositol 3-Kinases / Receptor, Platelet-Derived Growth Factor beta Limits: Animals / Humans Language: En Journal: Cancer Res Year: 2001 Document type: Article Affiliation country: Country of publication: