The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidation. | J Neuropathol Exp Neurol;61(4): 368-74, 2002 Apr. | MEDLINE

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The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidation.

Baes, M; Gressens, P; Huyghe, S; De, Nys K; Qi, C; Jia, Y; Mannaerts, G P; Evrard, P; Van, Veldhoven P P; Declercq, P E; Reddy, J K.

Affiliation

Baes M; Laboratory of Clinical Chemistry, Faculty of Pharmaceutical Sciences, KU Leuven, Belgium.

J Neuropathol Exp Neurol ; 61(4): 368-74, 2002 Apr.

Article in En | MEDLINE | ID: mdl-11939592

Subject(s)

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Collection: 01-internacional Database: MEDLINE Main subject: Zellweger Syndrome / Cell Movement / Receptors, Cytoplasmic and Nuclear / Peroxisomes / Neurons Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neuropathol Exp Neurol Year: 2002 Document type: Article Affiliation country: Country of publication:

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Collection: 01-internacional Database: MEDLINE Main subject: Zellweger Syndrome / Cell Movement / Receptors, Cytoplasmic and Nuclear / Peroxisomes / Neurons Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Neuropathol Exp Neurol Year: 2002 Document type: Article Affiliation country: Country of publication: