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Normalizing the gene dosage of Dyrk1A in a mouse model of Down syndrome rescues several Alzheimer's disease phenotypes.
García-Cerro, Susana; Rueda, Noemí; Vidal, Verónica; Lantigua, Sara; Martínez-Cué, Carmen.
Affiliation
  • García-Cerro S; Department of Anatomical Pathology, Pharmacology and Microbiology, Faculty of Medicine, University of Barcelona, Barcelona, Spain.
  • Rueda N; Department of Physiology and Pharmacology, Faculty of Medicine, University of Cantabria, Santander, Spain.
  • Vidal V; Department of Physiology and Pharmacology, Faculty of Medicine, University of Cantabria, Santander, Spain.
  • Lantigua S; Department of Physiology and Pharmacology, Faculty of Medicine, University of Cantabria, Santander, Spain.
  • Martínez-Cué C; Department of Physiology and Pharmacology, Faculty of Medicine, University of Cantabria, Santander, Spain. Electronic address: martinec@unican.es.
Neurobiol Dis ; 106: 76-88, 2017 Oct.
Article in En | MEDLINE | ID: mdl-28647555
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Protein-Tyrosine Kinases / Protein Serine-Threonine Kinases / Down Syndrome / Alzheimer Disease Limits: Animals Language: En Journal: Neurobiol Dis Journal subject: NEUROLOGIA Year: 2017 Document type: Article Affiliation country: Country of publication:
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Protein-Tyrosine Kinases / Protein Serine-Threonine Kinases / Down Syndrome / Alzheimer Disease Limits: Animals Language: En Journal: Neurobiol Dis Journal subject: NEUROLOGIA Year: 2017 Document type: Article Affiliation country: Country of publication: