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Nutritional Preconditioning of Apigenin Alleviates Myocardial Ischemia/Reperfusion Injury via the Mitochondrial Pathway Mediated by Notch1/Hes1.
Huang, Huang; Lai, Songqing; Luo, Yong; Wan, Qing; Wu, Qicai; Wan, Li; Qi, Wanghong; Liu, Jichun.
Affiliation
  • Huang H; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Lai S; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Luo Y; Jiangxi Provincial Key Laboratory of Women's Reproductive Health, Jiangxi Provincial Maternal and Child Health Hospital, Nanchang 330006, China.
  • Wan Q; Department of Pharmacy, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Wu Q; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Wan L; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Qi W; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
  • Liu J; Department of Cardiac Surgery, The First Affiliated Hospital of Nanchang University, Nanchang, Jiangxi 330006, China.
Oxid Med Cell Longev ; 2019: 7973098, 2019.
Article in En | MEDLINE | ID: mdl-31015891
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Myocardial Reperfusion Injury / Apigenin / Receptor, Notch1 / Transcription Factor HES-1 / Mitochondria Limits: Animals Language: En Journal: Oxid Med Cell Longev Journal subject: METABOLISMO Year: 2019 Document type: Article Affiliation country: Country of publication:
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Myocardial Reperfusion Injury / Apigenin / Receptor, Notch1 / Transcription Factor HES-1 / Mitochondria Limits: Animals Language: En Journal: Oxid Med Cell Longev Journal subject: METABOLISMO Year: 2019 Document type: Article Affiliation country: Country of publication: