Your browser doesn't support javascript.
loading
Denitrosylation of nNOS induced by cerebral ischemia-reperfusion contributes to nitrosylation of CaMKII and its inhibition of autophosphorylation in hippocampal CA1.
Yu, L-M; Zhang, T-Y; Yin, X-H; Yang, Q; Lu, F; Yan, J-Z; Li, C.
Affiliation
  • Zhang TY; Research Center of Biochemistry and Molecular Biology and Jiangsu Key Laboratory of Brain Disease Bioinformation, Xuzhou Medical University, Xuzhou, Jiangsu, China. lichong_xzhmu@sina.com.
Eur Rev Med Pharmacol Sci ; 23(17): 7674-7683, 2019 Sep.
Article in En | MEDLINE | ID: mdl-31539160
Subject(s)
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Reperfusion Injury / Nitric Oxide Synthase Type I / Calcium-Calmodulin-Dependent Protein Kinase Type 2 / CA1 Region, Hippocampal Limits: Animals Language: En Journal: Eur Rev Med Pharmacol Sci Journal subject: FARMACOLOGIA / TOXICOLOGIA Year: 2019 Document type: Article Country of publication:
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Reperfusion Injury / Nitric Oxide Synthase Type I / Calcium-Calmodulin-Dependent Protein Kinase Type 2 / CA1 Region, Hippocampal Limits: Animals Language: En Journal: Eur Rev Med Pharmacol Sci Journal subject: FARMACOLOGIA / TOXICOLOGIA Year: 2019 Document type: Article Country of publication: