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Anthracycline cardiotoxicity is exacerbated by global p38ß genetic ablation in a sexually dimorphic manner but unaltered by cardiomyocyte-specific p38α loss.
George, Sharon A; Kiss, Alexi; Trampel, Katy Anne; Obaid, Sofian N; Tang, Lichao; Efimov, Igor R; Efimova, Tatiana.
Affiliation
  • George SA; Department of Biomedical Engineering, Northwestern University, Chicago, Illinois, United States.
  • Kiss A; Department of Biomedical Engineering, George Washington University, Washington, District of Columbia, United States.
  • Trampel KA; Department of Anatomy and Cell Biology, George Washington University, Washington, District of Columbia, United States.
  • Obaid SN; George Washington Cancer Center, Washington, District of Columbia, United States.
  • Tang L; Department of Biomedical Engineering, Northwestern University, Chicago, Illinois, United States.
  • Efimov IR; Department of Biomedical Engineering, George Washington University, Washington, District of Columbia, United States.
  • Efimova T; Department of Biomedical Engineering, George Washington University, Washington, District of Columbia, United States.
Am J Physiol Heart Circ Physiol ; 325(5): H983-H997, 2023 11 01.
Article in En | MEDLINE | ID: mdl-37624097
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Myocytes, Cardiac / Cardiotoxicity Type of study: Prognostic_studies Limits: Animals Language: En Journal: Am J Physiol Heart Circ Physiol Journal subject: CARDIOLOGIA / FISIOLOGIA Year: 2023 Document type: Article Affiliation country: Country of publication:
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Myocytes, Cardiac / Cardiotoxicity Type of study: Prognostic_studies Limits: Animals Language: En Journal: Am J Physiol Heart Circ Physiol Journal subject: CARDIOLOGIA / FISIOLOGIA Year: 2023 Document type: Article Affiliation country: Country of publication: