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H2S serves as the immunoregulatory essence of apoptotic cell death.
Hine, Christopher; Ponti, András K; Cáliz-Molina, María Ángeles; Martín-Montalvo, Alejandro.
Affiliation
  • Hine C; Cleveland Clinic Lerner Research Institute, Cleveland, OH 44195, USA. Electronic address: hinec@ccf.org.
  • Ponti AK; Cleveland Clinic Lerner Research Institute, Cleveland, OH 44195, USA.
  • Cáliz-Molina MÁ; Cleveland Clinic Lerner Research Institute, Cleveland, OH 44195, USA; Andalusian Molecular Biology and Regenerative Medicine Centre-CABIMER, Universidad de Sevilla-CSIC-Universidad Pablo de Olavide, 41092 Seville, Spain.
  • Martín-Montalvo A; Andalusian Molecular Biology and Regenerative Medicine Centre-CABIMER, Universidad de Sevilla-CSIC-Universidad Pablo de Olavide, 41092 Seville, Spain; Biomedical Research Network on Diabetes and Related Metabolic Diseases-CIBERDEM, Instituto de Salud Carlos III, Madrid, Spain.
Cell Metab ; 36(1): 3-5, 2024 01 02.
Article in En | MEDLINE | ID: mdl-38171337
ABSTRACT
Apoptosis supports tissue homeostasis and prevents immune disorders by removing damaged and functionally aberrant cells. Here, Ou et al. utilized genetic, pharmacological, and proteomic approaches focused on sulfur amino acid catabolism to discover that hydrogen sulfide (H2S) release during apoptosis suppresses Th17 cell differentiation, thus providing therapeutic targets for autoimmune diseases.
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteomics / Hydrogen Sulfide Language: En Journal: Cell Metab Journal subject: METABOLISMO Year: 2024 Document type: Article Country of publication:
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteomics / Hydrogen Sulfide Language: En Journal: Cell Metab Journal subject: METABOLISMO Year: 2024 Document type: Article Country of publication: