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rs10924104 in the expression enhancer motif of CD58 confers susceptibility to human autoimmune diseases.
Hitomi, Yuki; Ueno, Kazuko; Aiba, Yoshihiro; Nishida, Nao; Kawai, Yosuke; Kawashima, Minae; Khor, Seik-Soon; Takada, Sanami; Iwabuchi, Chisato; Nagasaki, Masao; Tokunaga, Katsushi; Nakamura, Minoru.
Afiliação
  • Hitomi Y; Department of Human Genetics, Research Institute, National Center for Global Health and Medicine, 1-21-1 Toyama, Shinjuku-ku, Tokyo, 162-8655, Japan. yhitomi@ri.ncgm.go.jp.
  • Ueno K; Genome Medical Science Project, National Center for Global Health and Medicine, Tokyo, Japan.
  • Aiba Y; Clinical Research Center, National Hospital Organization (NHO) Nagasaki Medical Center, Omura, Japan.
  • Nishida N; Department of Genomic Function and Diversity, Medical Research Institute, Tokyo Medical and Dental University, Tokyo, Japan.
  • Kawai Y; Genome Medical Science Project, National Center for Global Health and Medicine, Tokyo, Japan.
  • Kawashima M; Database Center for Life Science, Research Organization of Information and Systems, Kashiwa, Japan.
  • Khor SS; Genome Medical Science Project, National Center for Global Health and Medicine, Tokyo, Japan.
  • Takada S; Singapore Centre for Environmental Life Sciences Engineering, Nanyang Technological University, Singapore, Singapore.
  • Iwabuchi C; Department of Human Genetics, Research Institute, National Center for Global Health and Medicine, 1-21-1 Toyama, Shinjuku-ku, Tokyo, 162-8655, Japan.
  • Nagasaki M; Department of Human Genetics, Research Institute, National Center for Global Health and Medicine, 1-21-1 Toyama, Shinjuku-ku, Tokyo, 162-8655, Japan.
  • Tokunaga K; Division of Biomedical Information Analysis, Medical Institute of Bioregulation, Kyushu University, Fukuoka, Japan.
  • Nakamura M; Genome Medical Science Project, National Center for Global Health and Medicine, Tokyo, Japan.
Hum Genet ; 143(1): 19-33, 2024 Jan.
Article em En | MEDLINE | ID: mdl-37994973
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Lúpus Eritematoso Sistêmico / Esclerose Múltipla Limite: Humans Idioma: En Revista: Hum Genet Ano de publicação: 2024 Tipo de documento: Article País de afiliação: Japão País de publicação: Alemanha
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Doenças Autoimunes / Lúpus Eritematoso Sistêmico / Esclerose Múltipla Limite: Humans Idioma: En Revista: Hum Genet Ano de publicação: 2024 Tipo de documento: Article País de afiliação: Japão País de publicação: Alemanha