Detalhe da pesquisa
1.
Influence of prenatal EGCG treatment and Dyrk1a dosage reduction on craniofacial features associated with Down syndrome.
Hum Mol Genet
; 25(22): 4856-4869, 2016 11 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28172997
2.
Rescue of the abnormal skeletal phenotype in Ts65Dn Down syndrome mice using genetic and therapeutic modulation of trisomic Dyrk1a.
Hum Mol Genet
; 24(20): 5687-96, 2015 Oct 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-26206885
3.
Non-trisomic homeobox gene expression during craniofacial development in the Ts65Dn mouse model of Down syndrome.
Am J Med Genet A
; 161A(8): 1866-74, 2013 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-23843306
4.
Embryonic and not maternal trisomy causes developmental attenuation in the Ts65Dn mouse model for Down syndrome.
Dev Dyn
; 239(6): 1645-53, 2010 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-20503361
5.
Abnormal mineralization of the Ts65Dn Down syndrome mouse appendicular skeleton begins during embryonic development in a Dyrk1a-independent manner.
Mech Dev
; 136: 133-42, 2015 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-25556111
6.
Disruption of bone development and homeostasis by trisomy in Ts65Dn Down syndrome mice.
Bone
; 48(2): 275-80, 2011 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-20870049