Detalhe da pesquisa
1.
Ryanodine Receptor Glycation Favors Mitochondrial Damage in the Senescent Heart.
Circulation
; 139(7): 949-964, 2019 02 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-30586718
2.
Evolution and bad prognostic value of advanced glycation end products after acute heart failure: relation with body composition.
Cardiovasc Diabetol
; 16(1): 115, 2017 09 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28915840
3.
Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.
Aging Cell
; 21(3): e13564, 2022 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-35233924
4.
Mitochondrial ROS and mitochondria-targeted antioxidants in the aged heart.
Free Radic Biol Med
; 167: 109-124, 2021 05 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33716106
5.
Enhancing Glycolysis Protects against Ischemia-Reperfusion Injury by Reducing ROS Production.
Metabolites
; 10(4)2020 Mar 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-32235559
6.
Cardiomyocyte ageing and cardioprotection: consensus document from the ESC working groups cell biology of the heart and myocardial function.
Cardiovasc Res
; 116(11): 1835-1849, 2020 09 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-32384145
7.
Cyclophilins A and B oppositely regulate renal tubular epithelial cell phenotype.
J Mol Cell Biol
; 12(7): 499-514, 2020 07 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-32162654
8.
Dicarbonyl stress and mitochondrial dysfunction in the aged heart.
Aging (Albany NY)
; 15(9): 3223-3225, 2023 05 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37130430