Detalhe da pesquisa
1.
Constitutively active CCK2 receptor splice variant increases Src-dependent HIF-1 alpha expression and tumor growth.
Oncogene
; 26(7): 1013-9, 2007 Feb 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-16909104
2.
Suppression of experimental autoimmune myasthenia gravis in IL-10 gene-disrupted mice is associated with reduced B cells and serum cytotoxicity on mouse cell line expressing AChR.
J Neuroimmunol
; 111(1-2): 152-60, 2000 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-11063833
3.
Lymphotoxin-alpha deficiency completely protects C57BL/6 mice from developing clinical experimental autoimmune myasthenia gravis.
J Neuroimmunol
; 113(1): 109-18, 2001 Feb 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-11137582
4.
Association of colony variation in Serratia marcescens with the differential expression of protease and type 1 fimbriae.
FEMS Microbiol Lett
; 133(1-2): 41-5, 1995 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-8566711
5.
Establishment of a mouse model of myasthenia gravis which mimics human myasthenia gravis pathogenesis for immune intervention.
Adv Exp Med Biol
; 383: 195-9, 1995.
Artigo
em Inglês
| MEDLINE | ID: mdl-8644502
6.
The pathogenic role of acetylcholine receptor alpha chain epitope within alpha 146-162 in the development of experimental autoimmune myasthenia gravis in C57BL6 mice.
Clin Immunol Immunopathol
; 73(3): 338-43, 1994 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-7955562
7.
Fas/Fas ligand pathway, apoptosis, and clonal anergy involved in systemic acetylcholine receptor T cell epitope tolerance.
J Immunol
; 166(5): 3458-67, 2001 Mar 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-11207304
8.
Experimental autoimmune myasthenia gravis in the mouse.
Curr Protoc Immunol
; Chapter 15: Unit 15.8, 2001 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-18432738
9.
Major histocompatibility complex class II gene disruption prevents experimental autoimmune myasthenia gravis.
J Immunol
; 152(6): 3152-7, 1994 Mar 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-8144909
10.
Tolerance to a dominant T cell epitope in the acetylcholine receptor molecule induces epitope spread and suppresses murine myasthenia gravis.
J Immunol
; 159(6): 3016-23, 1997 Sep 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-9300727
11.
TCR gene usage in experimental autoimmune myasthenia gravis pathogenesis. Usage of multiple TCRBV genes in the H-2b strains.
J Immunol
; 154(7): 3603-10, 1995 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-7897239
12.
IFN-alpha treatment suppresses the development of experimental autoimmune myasthenia gravis.
J Immunol
; 154(11): 6203-8, 1995 Jun 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-7751658
13.
IFN-alpha therapy is effective in suppressing the clinical experimental myasthenia gravis.
J Immunol
; 157(12): 5675-82, 1996 Dec 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-8955221
14.
Effect of MHC class I and CD8 cell deficiency on experimental autoimmune myasthenia gravis pathogenesis.
J Immunol
; 153(11): 5330-5, 1994 Dec 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-7963585
15.
The effect of B cell deficiency on the immune response to acetylcholine receptor and the development of experimental autoimmune myasthenia gravis.
Clin Immunol Immunopathol
; 87(3): 266-75, 1998 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-9646836
16.
HLA-DQ6 transgenic mice resistance to experimental autoimmune myasthenia gravis is linked to reduced acetylcholine receptor-specific IFN-gamma, IL-2 and IL-10 production.
J Autoimmun
; 17(3): 175-80, 2001 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-11712854
17.
[Trial application of transfection tests in feasibility studies for transforming DNA in white blood cells of patients with chronic lymphatic leukemia]. / Próba zastosowania testów transfekcyjnych w badaniach wlasciwosci transformujacych DNA z krwinek bialych chorych na przewlekla bialaczke limfatyczna.
Pol Tyg Lek
; 45(21-22): 412-6, 1990.
Artigo
em Polonês
| MEDLINE | ID: mdl-2267191
18.
Experimental autoimmune myasthenia gravis in B10.BV8S2 transgenic mice: preferential usage of TCRAV1 gene by lymphocytes responding to acetylcholine receptor.
J Immunol
; 158(12): 6006-12, 1997 Jun 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-9190955