Detalhe da pesquisa
1.
NOX5 expression is increased in intramyocardial blood vessels and cardiomyocytes after acute myocardial infarction in humans.
Am J Pathol
; 180(6): 2222-9, 2012 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-22503554
2.
Loss of DPP4 activity is related to a prothrombogenic status of endothelial cells: implications for the coronary microvasculature of myocardial infarction patients.
Basic Res Cardiol
; 107(1): 233, 2012 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-22167343
3.
Homocysteine induces phosphatidylserine exposure in cardiomyocytes through inhibition of Rho kinase and flippase activity.
Cell Physiol Biochem
; 28(1): 53-62, 2011.
Artigo
em Inglês
| MEDLINE | ID: mdl-21865848
4.
NOX2, p22phox and p47phox are targeted to the nuclear pore complex in ischemic cardiomyocytes colocalizing with local reactive oxygen species.
Cell Physiol Biochem
; 27(5): 471-8, 2011.
Artigo
em Inglês
| MEDLINE | ID: mdl-21691064
5.
Homocysteine-induced cardiomyocyte apoptosis and plasma membrane flip-flop are independent of S-adenosylhomocysteine: a crucial role for nuclear p47(phox).
Mol Cell Biochem
; 358(1-2): 229-39, 2011 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-21739151
6.
Intravenous clusterin administration reduces myocardial infarct size in rats.
Eur J Clin Invest
; 40(10): 893-902, 2010 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-20854280
7.
p47phox-Dependent Reactive Oxygen Species Stimulate Nuclear Translocation of the FoxO1 Transcription Factor During Metabolic Inhibition in Cardiomyoblasts.
Cell Biochem Biophys
; 76(3): 401-410, 2018 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-29956081
8.
Early NADPH oxidase-2 activation is crucial in phenylephrine-induced hypertrophy of H9c2 cells.
Cell Signal
; 26(9): 1818-24, 2014 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-24794531
9.
S-Adenosylhomocysteine induces apoptosis and phosphatidylserine exposure in endothelial cells independent of homocysteine.
Atherosclerosis
; 221(1): 48-54, 2012 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-22204864
10.
Prolonged C1 inhibitor administration improves local healing of burn wounds and reduces myocardial inflammation in a rat burn wound model.
J Burn Care Res
; 33(4): 544-51, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-22447201