RESUMO
We examined the seasonal cycle of radon concentration observed at King Sejong Station (KSG, 62°S), Antarctic Peninsula, during the period 2013-2016. The distribution of monthly radon concentration was found to be highly positively skewed from March through October (austral autumn to spring) due to large numbers of short-lived periods of high radon concentration. The global atmospheric chemistry model (CAM-Chem), which includes all global terrestrial sources of radon except for those in Antarctica, well reproduces the observed seasonal cycle of monthly-mean radon concentration at KSG. Further offline experiments suggest that uncertainties in radon emissions over South America and the Southern Ocean should be improved for the simulations of radon in Antarctica. The results demonstrate that seasonally varying transport of radon in the boundary layer from South America substantially affects the seasonality of monthly mean radon concentration at KSG. The composite analyses further reveal that high radon events at KSG are the result of a distinct east-west dipole-like structure associated with surface cyclonic circulation over the Bellingshausen Sea and anticyclonic circulation in the Weddell Sea. This atmospheric pattern provides favorable conditions for radon transport into KSG from the northwest. The relationship between radon concentration at KSG and climate variability is also discussed in this study.
Assuntos
Radônio , Regiões Antárticas , Clima , Estações do AnoRESUMO
This study analyzes the influence of the Pacific-Japan (PJ) atmospheric teleconnection pattern and its interaction with oceanic processes on sea surface warming over the Northwestern Pacific. The PJ pattern is a thermally driven Rossby wave that originates over the tropical western Pacific through deep convection and propagates toward high latitudes. It plays a significant role in sea surface warming by inducing anticyclonic circulation and the corresponding northwestward extension of the subtropical high over the Northwestern Pacific. This study revealed that the key processes responsible for sea surface warming were an increase in insolation and a decrease in the ocean-to-atmosphere latent heat flux under the anticyclonic conditions driven by the PJ. This finding provides valuable insights into the role of atmospheric processes, we refer to it as the "atmospheric pathway", in the development of East Asian marine heatwaves (MHWs). A detailed understanding of this process will contribute to the prediction and mitigation of MHWs in East Asian countries.
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Notable changes in the wintertime Arctic atmospheric circulation have occurred over the last few decades. Despite its importance in understanding the recent changes in the Northern Hemisphere midlatitude climate, it remains unclear whether and how these changes are affected by recent Arctic sea ice loss. In this study, a regional scale model is used to separate the direct sea ice influence from the natural variability of large-scale atmospheric circulation. Results show that, in response to sea ice loss, the increase of geopotential height in the mid-to-upper troposphere is robust across the simulations, but the magnitude of the response is highly dependent on the background state of the atmosphere. In most cases the sea ice loss-induced atmospheric warming is trapped near the surface due to the high vertical stability of winter Arctic lower troposphere, accordingly, resulting in a small response of geopotential height. However, when a low-pressure system is located over the Barents Sea, the relatively weak stability allows an upward transport of the surface warming, causing a significantly larger geopotential height increase. This strong state-dependence of atmospheric response which is also found in recent studies using global-scale model experiments, highlights the importance of accurately representing the atmospheric background state for numerical model assessments of sea ice influence.
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Recent Antarctic surface climate change has been characterized by greater warming trends in West Antarctica than in East Antarctica. Although this asymmetric feature is well recognized, its origin remains poorly understood. Here, by analyzing observation data and multimodel results, we show that a west-east asymmetric internal mode amplified in austral winter originates from the harmony of the atmosphere-ocean coupled feedback off West Antarctica and the Antarctic terrain. The warmer ocean temperature over the West Antarctic sector has positive feedback, with an anomalous upper-tropospheric anticyclonic circulation response centered over West Antarctica, in which the strength of the feedback is controlled by the Antarctic topographic layout and the annual cycle. The current west-east asymmetry of Antarctic surface climate change is undoubtedly of natural origin because no external factors (e.g., orbital or anthropogenic factors) contribute to the asymmetric mode.
RESUMO
In January 2016, the Arctic experienced an extremely anomalous warming event after an extraordinary increase in air temperature at the end of 2015. During this event, a strong intrusion of warm and moist air and an increase in downward longwave radiation, as well as a loss of sea ice in the Barents and Kara seas, were observed. Observational analyses revealed that the abrupt warming was triggered by the entry of a strong Atlantic windstorm into the Arctic in late December 2015, which brought enormous moist and warm air masses to the Arctic. Although the storm terminated at the eastern coast of Greenland in late December, it was followed by a prolonged blocking period in early 2016 that sustained the extreme Arctic warming. Numerical experiments indicate that the warming effect of sea ice loss and associated upward turbulent heat fluxes are relatively minor in this event. This result suggests the importance of the synoptically driven warm and moist air intrusion into the Arctic as a primary contributing factor of this extreme Arctic warming event.
RESUMO
BACKGROUND: AG60 is a complex of acriflavine and guanosine. Our previous study revealed that AG60 had not only in vitro antitumor activities in several human cancer cell lines, but also strong antitumor effects in animal experiments using p388 or S180 cells-implanted mice. METHODS: Antitumor effects of AG60 were compared with those of Adriamycin, acriflavine, guanosine or control group. Body weight, tumor weight change, and survival time were measured in Ehrlich carcinoma cells implanted ICR mice. RESULTS: Body weights in AG60, acriflavine, or Adriamycin treated groups were significantly lower than those in control group during 30 day observation period(p<0.05). The percent tumor growth inhibition of AG60, Adriamycin, acriflavine, or guanosine two weeks after last treatment was respectively 86% (T/C%=14), 83% (T/C%=17), 68%(T/C%=32), 41% (T/C%=59). According to above data, tumor growth inhibition in AG60 treated group was significantly stronger than that in control, acriflavine or guanosine treated group(p<0.01), but there was no significant difference between AG60 and Adriamycin treated group. Mean survival time in control, AG60, Adriamycin, acriflavine, or guanosine treated group was respectively 33+/-3.9 days, 68+/-4.2 days, 54+/-5.8 days, 36+/-3.8 days, 50+/-8.1 days. CONCLUSIONS: The anti-tumor effect of AG60 against Ehrlich tumor was significantly stronger than that of control, acriflavine or guanosine, and comparable with Adriamycin. Mean survival time in AG60 treated group was significantly longer than that in control, acrifavine, guanosine or Adriamysin treated group.