Detalhe da pesquisa
1.
Unprocessed genomic uracil as a source of DNA replication stress in cancer cells.
Mol Cell
; 84(11): 2036-2052.e7, 2024 Jun 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-38688279
2.
Tyrosyl-DNA phosphodiesterases: rescuing the genome from the risks of relaxation.
Nucleic Acids Res
; 46(2): 520-537, 2018 01 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-29216365
3.
TDP1 suppresses mis-joining of radiomimetic DNA double-strand breaks and cooperates with Artemis to promote optimal nonhomologous end joining.
Nucleic Acids Res
; 46(17): 8926-8939, 2018 09 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-30113698
4.
APOBEC3A induces DNA gaps through PRIMPOL and confers gap-associated therapeutic vulnerability.
Sci Adv
; 10(3): eadk2771, 2024 Jan 19.
Artigo
em Inglês
| MEDLINE | ID: mdl-38241374
5.
DNA binding and RAD51 engagement by the BRCA2 C-terminus orchestrate DNA repair and replication fork preservation.
Nat Commun
; 14(1): 432, 2023 01 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-36702902
6.
Bloom helicase mediates formation of large single-stranded DNA loops during DNA end processing.
Nat Commun
; 13(1): 2248, 2022 04 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-35473934
7.
Mechanism and significance of chromosome damage repair by homologous recombination.
Essays Biochem
; 64(5): 779-790, 2020 10 26.
Artigo
em Inglês
| MEDLINE | ID: mdl-32756864
8.
Tumor cell escape from therapy-induced senescence.
Biochem Pharmacol
; 162: 202-212, 2019 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-30576620
9.
Persistent 3'-phosphate termini and increased cytotoxicity of radiomimetic DNA double-strand breaks in cells lacking polynucleotide kinase/phosphatase despite presence of an alternative 3'-phosphatase.
DNA Repair (Amst)
; 68: 12-24, 2018 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-29807321