Detalhe da pesquisa
1.
Dual effect of cardiac FKBP12.6 overexpression on excitation-contraction coupling and the incidence of ventricular arrhythmia depending on its expression level.
J Mol Cell Cardiol
; 188: 15-29, 2024 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-38224852
2.
Cardiac Overexpression of PDE4B Blunts ß-Adrenergic Response and Maladaptive Remodeling in Heart Failure.
Circulation
; 142(2): 161-174, 2020 07 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-32264695
3.
Orai1 Channel Inhibition Preserves Left Ventricular Systolic Function and Normal Ca2+ Handling After Pressure Overload.
Circulation
; 141(3): 199-216, 2020 01 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-31906693
4.
Microtubule cytoskeleton regulates Connexin 43 localization and cardiac conduction in cardiomyopathy caused by mutation in A-type lamins gene.
Hum Mol Genet
; 28(24): 4043-4052, 2019 12 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-29893868
5.
Specific Activation of the Alternative Cardiac Promoter of Cacna1c by the Mineralocorticoid Receptor.
Circ Res
; 122(7): e49-e61, 2018 03 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-29467196
6.
Synergic PDE3 and PDE4 control intracellular cAMP and cardiac excitation-contraction coupling in a porcine model.
J Mol Cell Cardiol
; 133: 57-66, 2019 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-31158360
7.
Two-pore channels (TPCs) acts as a hub for excitation-contraction coupling, metabolism and cardiac hypertrophy signalling.
Cell Calcium
; 117: 102839, 2024 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-38134531
8.
Phosphodiesterases type 2, 3 and 4 promote vascular tone in mesenteric arteries from rats with heart failure.
Eur J Pharmacol
; 944: 175562, 2023 Apr 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-36736940
9.
EPAC1 inhibition protects the heart from doxorubicin-induced toxicity.
Elife
; 122023 08 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-37551870
10.
CD38-NADase is a new major contributor to Duchenne muscular dystrophic phenotype.
EMBO Mol Med
; 14(5): e12860, 2022 05 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-35298089
11.
Decreased expression and activity of cAMP phosphodiesterases in cardiac hypertrophy and its impact on beta-adrenergic cAMP signals.
Circ Res
; 105(8): 784-92, 2009 Oct 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-19745166
12.
Activation of sarcolipin expression and altered calcium cycling in LMNA cardiomyopathy.
Biochem Biophys Rep
; 22: 100767, 2020 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-32490213
13.
Contribution of BKCa channels to vascular tone regulation by PDE3 and PDE4 is lost in heart failure.
Cardiovasc Res
; 115(1): 130-144, 2019 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-29939224
14.
A specific pattern of phosphodiesterases controls the cAMP signals generated by different Gs-coupled receptors in adult rat ventricular myocytes.
Circ Res
; 98(8): 1081-8, 2006 Apr 28.
Artigo
em Inglês
| MEDLINE | ID: mdl-16556871
15.
PDE4 and mAKAPß are nodal organizers of ß2-ARs nuclear PKA signalling in cardiac myocytes.
Cardiovasc Res
; 114(11): 1499-1511, 2018 09 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-29733383
16.
Standard and Strain Measurements by Echocardiography Detect Early Overloaded Right Ventricular Dysfunction: Validation against Hemodynamic and Myocyte Contractility Changes in a Large Animal Model.
J Am Soc Echocardiogr
; 30(11): 1138-1147.e4, 2017 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-28864150
17.
Calmodulin kinase II inhibition limits the pro-arrhythmic Ca2+ waves induced by cAMP-phosphodiesterase inhibitors.
Cardiovasc Res
; 110(1): 151-61, 2016 May 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-26851245
18.
Reconciling depressed Ca2+ sparks occurrence with enhanced RyR2 activity in failing mice cardiomyocytes.
J Gen Physiol
; 146(4): 295-306, 2015 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-26371209
19.
Sexual dimorphism of doxorubicin-mediated cardiotoxicity: potential role of energy metabolism remodeling.
Circ Heart Fail
; 8(1): 98-108, 2015 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-25420486
20.
Control of cytoplasmic and nuclear protein kinase A by phosphodiesterases and phosphatases in cardiac myocytes.
Cardiovasc Res
; 102(1): 97-106, 2014 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-24550350