Assuntos
Antígenos de Plantas/química , Antígenos de Plantas/imunologia , Epitopos de Linfócito B/química , Epitopos de Linfócito B/imunologia , Glicoproteínas/química , Glicoproteínas/imunologia , Imunoglobulina E/imunologia , Hipersensibilidade a Amendoim/imunologia , Proteínas de Plantas/química , Proteínas de Plantas/imunologia , Adulto , Humanos , Masculino , Proteínas de Membrana , Hipersensibilidade a Amendoim/complicações , Conformação Proteica , Prurido/etiologiaRESUMO
Heterotopic heart transplantation is an alternative option in end-stage heart disease. We present a patient with a heterotopic allograft that maintained hemodynamics at the period of a malignant ventricular dysrhythmia and supports the notion that these otherwise lethal dysrhythmias of the native heart may not carry the same risk of sudden heart death in patients with heterotopic heart transplantation.
Assuntos
Eletrocardiografia , Transplante de Coração/fisiologia , Hemodinâmica/fisiologia , Taquicardia/fisiopatologia , Transplante Heterotópico/fisiologia , Humanos , Masculino , Pessoa de Meia-Idade , Taquicardia/diagnóstico , Taquicardia/terapiaRESUMO
Left coronary artery arising from the right sinus of Valsalva is a rare congenital coronary anomaly. This anomaly is either benign or serious, depending on the relation of the anomalous left coronary artery to the aorta and pulmonary artery. Potentially serious anomaly is associated with sudden cardiac death and warrants prophylactic coronary bypass surgery. A rare case of anomalous left coronary artery arising from the right sinus of Valsalva is reported, documented by coronary angiography; however, it took a safer course between the aorta and pulmonary artery. Prophylactic surgery was not performed, for this benign anomaly may not carry the same risk of sudden cardiac death.
Assuntos
Anomalias dos Vasos Coronários/diagnóstico , Seio Aórtico/anormalidades , Adulto , Aorta/patologia , Angiografia Coronária , Ponte de Artéria Coronária , Anomalias dos Vasos Coronários/patologia , Morte Súbita Cardíaca/prevenção & controle , Feminino , Humanos , Artéria Pulmonar/patologia , Seio Aórtico/patologiaRESUMO
Anticoagulation in the form of intravenous heparin is used after coronary angioplasty to prevent thrombosis. Ancrod, a rapid-acting defibrinogenating agent, has been used in various clinical settings that require anticoagulation. We present the use of ancrod after percutaneous transluminal coronary angioplasty in a patients with heparin-induced thrombopathia.
Assuntos
Ancrod/uso terapêutico , Angioplastia Coronária com Balão , Anticoagulantes/uso terapêutico , Idoso , Anticoagulantes/efeitos adversos , Transtornos da Coagulação Sanguínea/induzido quimicamente , Transtornos da Coagulação Sanguínea/tratamento farmacológico , Feminino , Heparina/efeitos adversos , HumanosRESUMO
Papillary fibroelastomas are rare, benign tumors of the heart that often are incidental findings at autopsy or cardiac surgery, or during echocardiography. These tumors are predisposed to valvular involvement, and all such lesions detected by echocardiography have occurred in the left side of the heart, except for 3 on the tricuspid valve. Left-sided lesions have been associated with embolic phenomena; however, embolization of right-sided lesions has never been documented. We report a tricuspid valve fibroelastoma detected by 2-dimensional and transesophageal echocardiography, and confirmed by surgical excision and biopsy. To our knowledge, this is the 1st reported case of pulmonary embolization of papillary fibroelastoma arising from the tricuspid valve.
RESUMO
Awareness of pathophysiology and differential diagnosis of this entity is crucial for management. Edema may be ipsilateral or contralateral to the perfusion defect, or may be variable. A variable case is presented.
Assuntos
Edema Pulmonar/diagnóstico , Adulto , Diagnóstico Diferencial , Epilepsia/complicações , Feminino , Humanos , Edema Pulmonar/etiologia , Edema Pulmonar/terapiaRESUMO
Acute dissections mandate surgery after the patient has been stabilized medically. Hypertension appears to be a prerequisite in the pathogenesis of aortic dissection, and control of ventricular contraction velocity may be prophylactic.