Detalhe da pesquisa
1.
Mechanistic Insights of the LEMD2 p.L13R Mutation and Its Role in Cardiomyopathy.
Circ Res
; 132(2): e43-e58, 2023 01 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-36656972
2.
Activation of the integrated stress response rewires cardiac metabolism in Barth syndrome.
Basic Res Cardiol
; 118(1): 47, 2023 11 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-37930434
3.
An arrhythmogenic metabolite in atrial fibrillation.
J Transl Med
; 21(1): 566, 2023 08 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-37620858
4.
Regulation of titin-based cardiac stiffness by unfolded domain oxidation (UnDOx).
Proc Natl Acad Sci U S A
; 117(39): 24545-24556, 2020 09 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-32929035
5.
Loss of Mitochondrial Ca2+ Uniporter Limits Inotropic Reserve and Provides Trigger and Substrate for Arrhythmias in Barth Syndrome Cardiomyopathy.
Circulation
; 144(21): 1694-1713, 2021 11 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-34648376
6.
Tachycardiomyopathy entails a dysfunctional pattern of interrelated mitochondrial functions.
Basic Res Cardiol
; 117(1): 45, 2022 09 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-36068416
7.
Cathepsin A contributes to left ventricular remodeling by degrading extracellular superoxide dismutase in mice.
J Biol Chem
; 295(36): 12605-12617, 2020 09 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-32647007
8.
CaMKII does not control mitochondrial Ca2+ uptake in cardiac myocytes.
J Physiol
; 598(7): 1361-1376, 2020 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-30770570
9.
Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart.
Basic Res Cardiol
; 115(5): 53, 2020 08 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-32748289
10.
Inhibition of MicroRNA-146a and Overexpression of Its Target Dihydrolipoyl Succinyltransferase Protect Against Pressure Overload-Induced Cardiac Hypertrophy and Dysfunction.
Circulation
; 136(8): 747-761, 2017 Aug 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-28611091
11.
Raf kinase inhibitor protein mediates myocardial fibrosis under conditions of enhanced myocardial oxidative stress.
Basic Res Cardiol
; 113(6): 42, 2018 09 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-30191336
12.
Myocardial infarction is sufficient to increase GLP-1 secretion, leading to improved left ventricular contractility and mitochondrial respiratory capacity.
Diabetes Obes Metab
; 20(12): 2911-2918, 2018 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-30033664
13.
Low STAT3 expression sensitizes to toxic effects of ß-adrenergic receptor stimulation in peripartum cardiomyopathy.
Eur Heart J
; 38(5): 349-361, 2017 02 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-28201733
14.
Mitochondrial energetics and calcium coupling in the heart.
J Physiol
; 595(12): 3753-3763, 2017 06 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28105746
15.
Endogenous nitric oxide formation in cardiac myocytes does not control respiration during ß-adrenergic stimulation.
J Physiol
; 595(12): 3781-3798, 2017 06 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28229450
16.
Mitochondrial reactive oxygen species production and elimination.
J Mol Cell Cardiol
; 73: 26-33, 2014 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-24657720
17.
Effects of NPY-2 Receptor Antagonists, Semaglutide, PYY3-36, and Empagliflozin on Early MASLD in Diet-Induced Obese Rats.
Nutrients
; 16(6)2024 Mar 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-38542814
18.
GLP-1 and PYY for the treatment of obesity: a pilot study on the use of agonists and antagonists in diet-induced rats.
Endocr Connect
; 13(3)2024 Mar 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-38300808
19.
Epigenetic modulators link mitochondrial redox homeostasis to cardiac function in a sex-dependent manner.
Nat Commun
; 15(1): 2358, 2024 Mar 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-38509128
20.
Mutations in DNAJC19 cause altered mitochondrial structure and increased mitochondrial respiration in human iPSC-derived cardiomyocytes.
Mol Metab
; 79: 101859, 2024 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-38142971