Detalhe da pesquisa
1.
Combining fibril-induced alpha-synuclein aggregation and 6-hydroxydopamine in a mouse model of Parkinson's disease and the effect of cerebral dopamine neurotrophic factor on the induced neurodegeneration.
Eur J Neurosci
; 59(1): 132-153, 2024 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-38072889
2.
Elevated endogenous GDNF induces altered dopamine signalling in mice and correlates with clinical severity in schizophrenia.
Mol Psychiatry
; 27(8): 3247-3261, 2022 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-35618883
3.
Cerebral dopamine neurotrophic factor reduces α-synuclein aggregation and propagation and alleviates behavioral alterations in vivo.
Mol Ther
; 29(9): 2821-2840, 2021 09 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-33940158
4.
Soluble CD163 Changes Indicate Monocyte Association With Cognitive Deficits in Parkinson's Disease.
Mov Disord
; 36(4): 963-976, 2021 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-33332647
5.
Cerebral dopamine neurotrophic factor-deficiency leads to degeneration of enteric neurons and altered brain dopamine neuronal function in mice.
Neurobiol Dis
; 134: 104696, 2020 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-31783118
6.
GDNF/RET Signaling Pathway Activation Eliminates Lewy Body Pathology in Midbrain Dopamine Neurons.
Mov Disord
; 35(12): 2279-2289, 2020 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-32964492
7.
Correction: GDNF Overexpression from the Native Locus Reveals its Role in the Nigrostriatal Dopaminergic System Function.
PLoS Genet
; 12(1): e1005808, 2016 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-26752407
8.
Dampened Amphetamine-Stimulated Behavior and Altered Dopamine Transporter Function in the Absence of Brain GDNF.
J Neurosci
; 37(6): 1581-1590, 2017 02 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-28096470
9.
GDNF Overexpression from the Native Locus Reveals its Role in the Nigrostriatal Dopaminergic System Function.
PLoS Genet
; 11(12): e1005710, 2015 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-26681446
10.
Changes in neuronal dopamine homeostasis following 1-methyl-4-phenylpyridinium (MPP+) exposure.
J Biol Chem
; 290(11): 6799-809, 2015 Mar 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-25596531
11.
GABA site agonist gaboxadol induces addiction-predicting persistent changes in ventral tegmental area dopamine neurons but is not rewarding in mice or baboons.
J Neurosci
; 32(15): 5310-20, 2012 Apr 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-22496576
12.
Feasibility of combining alpha-synuclein aggregation and 6-OHDA in embryonic midbrain culture for modeling dopamine neuron degeneration.
Neurosci Lett
; 816: 137510, 2023 11 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37802418
13.
Excessive novelty-induced c-Fos expression and altered neurogenesis in the hippocampus of GluA1 knockout mice.
Eur J Neurosci
; 33(1): 161-74, 2011 Jan.
Artigo
em Inglês
| MEDLINE | ID: mdl-21073553
14.
ADHD-like behaviors caused by inactivation of a transcription factor controlling the balance of inhibitory and excitatory neuron development in the mouse anterior brainstem.
Transl Psychiatry
; 10(1): 357, 2020 10 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-33087695
15.
Conditioned Aversion and Neuroplasticity Induced by a Superagonist of Extrasynaptic GABAA Receptors: Correlation With Activation of the Oval BNST Neurons and CRF Mechanisms.
Front Mol Neurosci
; 12: 130, 2019.
Artigo
em Inglês
| MEDLINE | ID: mdl-31178693
16.
Prolyl Oligopeptidase Regulates Dopamine Transporter Phosphorylation in the Nigrostriatal Pathway of Mouse.
Mol Neurobiol
; 55(1): 470-482, 2018 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-27966077
17.
GABAA receptor drugs and neuronal plasticity in reward and aversion: focus on the ventral tegmental area.
Front Pharmacol
; 5: 256, 2014.
Artigo
em Inglês
| MEDLINE | ID: mdl-25505414
18.
Evidence for a role of inhibition of orexinergic neurons in the anxiolytic and sedative effects of diazepam: A c-Fos study.
Pharmacol Biochem Behav
; 101(1): 115-24, 2012 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-22210490
19.
Importance of GluA1 subunit-containing AMPA glutamate receptors for morphine state-dependency.
PLoS One
; 7(5): e38325, 2012.
Artigo
em Inglês
| MEDLINE | ID: mdl-22675452