Detalhe da pesquisa
1.
ICOS:ICOS-ligand interaction is required for type 2 innate lymphoid cell function, homeostasis, and induction of airway hyperreactivity.
Immunity
; 42(3): 538-51, 2015 Mar 17.
Artigo
em Inglês
| MEDLINE | ID: mdl-25769613
2.
A GWAS approach identifies Dapp1 as a determinant of air pollution-induced airway hyperreactivity.
PLoS Genet
; 15(12): e1008528, 2019 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-31869344
3.
Type 2 innate lymphoid cell suppression by regulatory T cells attenuates airway hyperreactivity and requires inducible T-cell costimulator-inducible T-cell costimulator ligand interaction.
J Allergy Clin Immunol
; 139(5): 1468-1477.e2, 2017 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-27717665
4.
Nucleotide modifications enable rational design of TLR7-selective ligands by blocking RNase cleavage.
J Exp Med
; 221(2)2024 Feb 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-38095631
5.
An Engineered IL15 Cytokine Mutein Fused to an Anti-PD1 Improves Intratumoral T-cell Function and Antitumor Immunity.
Cancer Immunol Res
; 9(10): 1141-1157, 2021 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-34376502
6.
Exploiting MCL1 Dependency with Combination MEK + MCL1 Inhibitors Leads to Induction of Apoptosis and Tumor Regression in KRAS-Mutant Non-Small Cell Lung Cancer.
Cancer Discov
; 8(12): 1598-1613, 2018 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-30254092
7.
Deficiency in the nuclear-related factor erythroid 2 transcription factor (Nrf1) leads to genetic instability.
FEBS J
; 279(22): 4121-30, 2012 Nov.
Artigo
em Inglês
| MEDLINE | ID: mdl-22971132