Detalhe da pesquisa
1.
Minor intron splicing is critical for survival of lethal prostate cancer.
Mol Cell
; 83(12): 1983-2002.e11, 2023 Jun 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-37295433
2.
De Novo Variants in SPOP Cause Two Clinically Distinct Neurodevelopmental Disorders.
Am J Hum Genet
; 106(3): 405-411, 2020 03 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-32109420
3.
SPOP-mutant prostate cancer: Translating fundamental biology into patient care.
Cancer Lett
; 529: 11-18, 2022 03 31.
Artigo
em Inglês
| MEDLINE | ID: mdl-34974131
4.
Dynamic prostate cancer transcriptome analysis delineates the trajectory to disease progression.
Nat Commun
; 12(1): 7033, 2021 12 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-34857732
5.
Dual functions of SPOP and ERG dictate androgen therapy responses in prostate cancer.
Nat Commun
; 12(1): 734, 2021 02 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-33531470
6.
Opposing effects of cancer-type-specific SPOP mutants on BET protein degradation and sensitivity to BET inhibitors.
Nat Med
; 23(9): 1046-1054, 2017 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-28805821
7.
Morphologic changes and altered gene expression in an epithelioid hemangioendothelioma during a ten-year course of disease.
Pathol Res Pract
; 199(3): 165-70, 2003.
Artigo
em Inglês
| MEDLINE | ID: mdl-12812318
8.
Prostate cancer. Ubiquitylome analysis identifies dysregulation of effector substrates in SPOP-mutant prostate cancer.
Science
; 346(6205): 85-89, 2014 Oct 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-25278611