Detalhe da pesquisa
1.
Persistent myelin abnormalities in a third trimester-equivalent mouse model of fetal alcohol spectrum disorder.
Alcohol Clin Exp Res
; 46(1): 77-86, 2022 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-34825395
2.
Resistance of Postnatal Hippocampal Neurogenesis to Alcohol Toxicity in a Third Trimester-Equivalent Mouse Model of Gestational Alcohol Exposure.
Alcohol Clin Exp Res
; 43(12): 2504-2513, 2019 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-31573091
3.
Acute oligodendrocyte loss with persistent white matter injury in a third trimester equivalent mouse model of fetal alcohol spectrum disorder.
Glia
; 65(8): 1317-1332, 2017 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-28518477
4.
Ethanol exposure during the third trimester equivalent does not affect GABAA or AMPA receptor-mediated spontaneous synaptic transmission in rat CA3 pyramidal neurons.
J Negat Results Biomed
; 14: 19, 2015 Dec 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-26627643
5.
Prenatal ethanol exposure persistently impairs NMDA receptor-dependent activation of extracellular signal-regulated kinase in the mouse dentate gyrus.
J Neurochem
; 109(5): 1311-23, 2009 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-19317851
6.
Moderate Alcohol Exposure during the Rat Equivalent to the Third Trimester of Human Pregnancy Alters Regulation of GABAA Receptor-Mediated Synaptic Transmission by Dopamine in the Basolateral Amygdala.
Front Pediatr
; 2: 46, 2014.
Artigo
em Inglês
| MEDLINE | ID: mdl-24904907
7.
AMPAR-mediated synaptic transmission in the CA1 hippocampal region of neonatal rats: unexpected resistance to repeated ethanol exposure.
Alcohol
; 43(8): 619-25, 2009 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-20004339