Detalhe da pesquisa
1.
The HDAC9-associated risk locus promotes coronary artery disease by governing TWIST1.
PLoS Genet
; 18(6): e1010261, 2022 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-35714152
2.
PHACTR1 Is a Genetic Susceptibility Locus for Fibromuscular Dysplasia Supporting Its Complex Genetic Pattern of Inheritance.
PLoS Genet
; 12(10): e1006367, 2016 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-27792790
3.
FOXO3-mTOR metabolic cooperation in the regulation of erythroid cell maturation and homeostasis.
Am J Hematol
; 89(10): 954-63, 2014 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-24966026
4.
Genome-wide association meta-analysis of spontaneous coronary artery dissection identifies risk variants and genes related to artery integrity and tissue-mediated coagulation.
Nat Genet
; 55(6): 964-972, 2023 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-37248441
5.
Integrative Prioritization of Causal Genes for Coronary Artery Disease.
Circ Genom Precis Med
; 15(1): e003365, 2022 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-34961328
6.
Histone deacetylase 9 promotes endothelial-mesenchymal transition and an unfavorable atherosclerotic plaque phenotype.
J Clin Invest
; 131(15)2021 08 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-34338228
7.
Genetic investigation of fibromuscular dysplasia identifies risk loci and shared genetics with common cardiovascular diseases.
Nat Commun
; 12(1): 6031, 2021 10 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-34654805
8.
Rare loss-of-function mutations of PTGIR are enriched in fibromuscular dysplasia.
Cardiovasc Res
; 117(4): 1154-1165, 2021 03 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-32531060
9.
Tissue-Resident PDGFRα+ Progenitor Cells Contribute to Fibrosis versus Healing in a Context- and Spatiotemporally Dependent Manner.
Cell Rep
; 30(2): 555-570.e7, 2020 01 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-31940496
10.
A plasma proteogenomic signature for fibromuscular dysplasia.
Cardiovasc Res
; 116(1): 63-77, 2020 01 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-31424497
11.
Association of the PHACTR1/EDN1 Genetic Locus With Spontaneous Coronary Artery Dissection.
J Am Coll Cardiol
; 73(1): 58-66, 2019 01 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-30621952
12.
CD90 Identifies Adventitial Mesenchymal Progenitor Cells in Adult Human Medium- and Large-Sized Arteries.
Stem Cell Reports
; 11(1): 242-257, 2018 07 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-30008326
13.
Author Correction: Genetic investigation of fibromuscular dysplasia identifies risk loci and shared genetics with common cardiovascular diseases.
Nat Commun
; 13(1): 2251, 2022 Apr 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-35443759
14.
Matricellular Protein CCN5 Reverses Established Cardiac Fibrosis.
J Am Coll Cardiol
; 67(13): 1556-1568, 2016 04 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-27150688
15.
Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability.
Nat Commun
; 7: 11853, 2016 06 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-27340017
16.
Coronary artery manifestations of fibromuscular dysplasia.
J Am Coll Cardiol
; 64(10): 1033-46, 2014 Sep 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-25190240
17.
Corrigendum: Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability.
Nat Commun
; 8: 14710, 2017 02 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-28205517
18.
The anaplastic lymphoma kinase is an effective oncoantigen for lymphoma vaccination.
Nat Med
; 14(6): 676-80, 2008 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-18469826
19.
Ablation of oncogenic ALK is a viable therapeutic approach for anaplastic large-cell lymphomas.
Blood
; 107(2): 689-97, 2006 Jan 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-16189272