β-arrestin1 Promotes the Concentration of Mitochondrial Reactive Oxygen in T-ALL Cells via MiR-652-5p
Hao-Biao WANG; Shan LIU; Wei GUO; Hai-Yan LIU; Jie YU; Lin ZOU.
Journal of Experimental Hematology
; (6): 1456-1461, 2021.
Artigo em Zh | WPRIM | ID: wpr-922279
Documentos relacionados
[ß-arrestin1 Promotes the Concentration of Mitochondrial Reactive Oxygen in T-ALL Cells via MiR-652-5p].
Biasing Neurotensin Receptor Signaling to Arrest Psychostimulant Abuse.
miR-22-3p in the rostral ventrolateral medulla promotes hypertension through inhibiting ß-arrestin-1.
ß-Arrestin-independent endosomal cAMP signaling by a polypeptide hormone GPCR.
Structure of the neurotensin receptor 1 in complex with ß-arrestin 1.
In silico identification of a ß<sub>2</sub>-adrenoceptor allosteric site that selectively augments canonical ß<sub>2</sub>AR-Gs signaling and function.
GPCR-mediated ß-arrestin activation deconvoluted with single-molecule precision.
ß-arrestin1 is an E3 ubiquitin ligase adaptor for substrate linear polyubiquitination.
Beta-Arrestin1 Prevents Preeclampsia by Downregulation of Mechanosensitive AT1-B2 Receptor Heteromers.
Tail engagement of arrestin at the glucagon receptor.