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Nuclear 82-kDa choline acetyltransferase decreases amyloidogenic APP metabolism in neurons from APP/PS1 transgenic mice.
Albers, Shawn; Inthathirath, Fatima; Gill, Sandeep K; Winick-Ng, Warren; Jaworski, Ewa; Wong, Daisy Y L; Gros, Robert; Rylett, R Jane.
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  • Albers S; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Inthathirath F; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Gill SK; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Winick-Ng W; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Jaworski E; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Wong DY; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Gros R; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada.
  • Rylett RJ; Department of Physiology and Pharmacology, Schulich School of Medicine & Dentistry, University of Western Ontario, Molecular Medicine Research Group, Robarts Research Institute, London, Ontario, Canada. Electronic address: jane.rylett@schulich.uwo.ca.
Neurobiol Dis ; 69: 32-42, 2014 Sep.
Article em En | MEDLINE | ID: mdl-24844149
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Tipo de estudo: Prognostic_studies Limite: Animals / Humans Idioma: En Ano de publicação: 2014 Tipo de documento: Article