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Hemodialysis-induced Myocardial Stunning with No Obstructive Coronary Artery Disease: Pathophysiological Significance of Coronary Microvascular Dysfunction.
Yokoi, Masashi; Ito, Tsuyoshi; Suzuki, Wataru; Yamamoto, Junki; Nakayama, Takafumi; Ichihashi, Taku; Kikuchi, Shohei; Kitada, Shuichi; Goto, Toshihiko; Seo, Yoshihiro.
Afiliação
  • Yokoi M; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Ito T; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Suzuki W; Department of Cardiology, Aichi Medical University, Japan.
  • Yamamoto J; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Nakayama T; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Ichihashi T; Department of Cardiology, Nagoya City University East Medical Center, Japan.
  • Kikuchi S; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Kitada S; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Goto T; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
  • Seo Y; Department of Cardiology, Nagoya City University Graduate School of Medical Sciences, Japan.
Intern Med ; 2023 Dec 04.
Article em En | MEDLINE | ID: mdl-38044153
ABSTRACT
Hemodialysis (HD)-induced myocardial stunning, characterized by transient left ventricular systolic dysfunction during HD, has been reported to be common and associated with a poor prognosis. However, the pathophysiology is not fully understood. We herein report a case of HD-induced myocardial stunning without obstructive coronary artery disease complicated by coronary microvascular dysfunction (CMD), suggesting that CMD plays a crucial role in the pathophysiology of this disease.
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Idioma: En Ano de publicação: 2023 Tipo de documento: Article