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Kaku Igaku ; 45(4): 357-60, 2008 Nov.
Artículo en Japonés | MEDLINE | ID: mdl-19591408

RESUMEN

For the patients under long-term tube feeding, copper deficiency is known to be a cause of neutropenia and/or anemia. It is recognized as reversible myelodysplasia, since the condition improves by giving a copper supplementation. Myelodysplasia caused by copper deficiency is difficult to be diagnosed because it is not so common, and often it takes a long time to reach correct diagnosis. We reported usefulness of FDG-PET for the diagnosis of myelodysplasia caused by copper deficiency in early stage. The case was 46 y.o. male patient in vegetative state for 2 years after traumatic brain injury. Laboratory examination revealed slight leukopenia. PET/CT demonstrated high and diffuse FDG accumulation mainly in the vertebral bone marrow. Based on the lower levels of serum copper and ceruloprasmin, the patient was diagnosed as copper deficiency. After treatment of copper supplementation, FDG accumulation of the bone marrow disappeared, and the serum copper level has normalized. From the FDG-PET findings, even in the early stage of copper deficiency, high glucose metabolism of bone marrow was shown.


Asunto(s)
Cobre/deficiencia , Fluorodesoxiglucosa F18 , Defectos del Tubo Neural/diagnóstico por imagen , Defectos del Tubo Neural/etiología , Tomografía de Emisión de Positrones , Radiofármacos , Médula Ósea/metabolismo , Cobre/administración & dosificación , Diagnóstico Diferencial , Diagnóstico Precoz , Nutrición Enteral/efectos adversos , Glucosa/metabolismo , Humanos , Masculino , Persona de Mediana Edad , Defectos del Tubo Neural/tratamiento farmacológico
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